Parameter Copper  Sample 5 mL - blood - serum tube   Turnaround time 2 to 5 working days

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Parameter Culture of the uterus  Sample Uterine swab in special culture transport system Turnaround time 1-15 day for bacterial growth Several days (>15) for yeast and other fungal growth 

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Parameter Entails a detailed examination of cells, using advanced techniques not only to identify abnormalities and diagnose diseases but also to perform precise cell counts, including differential counts to distinguish between different cell types or identify specific cellular abnormalities. Sample Typically involves cell samples from tissues, lavages or fluid aspirations, with cell counts performed using staining techniques to facilitate easier observation and counting, and  to provide detailed counts and information on cell types. Turnaround time The results for advanced cytological studies, including differential cell counts, may take longer than basic cytology due to the complexity of the tests and analyses involved are usually ready within 2-4 working days.

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Shopping for someone else but not sure what to give them? Is he/she a Horse-lover ? Give them the gift of choice with a Equigerminal gift card. Gift cards are delivered by email and contain instructions to redeem them at checkout.Our gift cards have no additional processing fees.

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Our Equine Piroplasmosis qPCR Test provides accurate detection of the genome of Babesia caballi and Theileria equi, the primary agents responsible for Equine Piroplasmosis. This test is ideal for early infection detection and during febrile peaks. Product Overview qPCR Test: Detects the genome of Babesia caballi and Theileria equi, providing highly sensitive detection of piroplasms. Sample Requirements 5 mL of blood - collect blood in K3 EDTA tube for qPCR Turnaround Time Standard Processing: Results in 2 working days after sample arrival at the laboratory. Clients are responsible for organizing and covering the costs of sending the samples to the laboratory. What is Piroplasmosis? Equine piroplasmosis (EP) is a tick-borne disease of horses caused by the intraerythrocytic protozoan parasites Babesia caballi and Theileria equi. These agents are transmitted through a tick vector. Infected animals may remain carriers of these blood parasites for long periods and act as sources of infection for other ticks. The parasites are found in southern Europe, Asia, countries of the Commonwealth of Independent States, Africa, Cuba, South and Central America, and parts of the southern United States. Clinical Signs Incubation period: 12 to 19 days for T. equi and 10 to 30 days for B. caballi. Per-acute form: Rare, with only clinical observation being moribund or dead animals. Acute form: Fever, reduced appetite, elevated respiratory and pulse rates, congestion of mucous membranes, dark red urine, smaller and drier faecal balls, anaemia, and/or icterus. Subacute form: Similar to acute form but with weight loss and intermittent fever. Mucous membranes may vary from pale pink to bright yellow. Chronic form: Mild inappetence, poor performance, weight loss. Documented case fatality rates vary from 10–50%. Transmission Babesia caballi sporozoites invade red blood cells (RBCs), transforming into trophozoites which divide into merozoites, capable of infecting new RBCs. B. caballi can be found in various organs of tick vectors and transmit transovarially. Theileria equi sporozoites, inoculated into horses via a tick bite, invade lymphocytes, develop into schizonts, and release merozoites that invade RBCs. T. equi develop in the salivary glands of the tick vector and are not transmitted transovarially. Transmission is also possible through mechanical vectors contaminated by infected blood. Prevention Sanitary Prophylaxis: Testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals. Medical Prophylaxis: No current biological products are available. Antiprotozoal agents only temporarily clear T. equi from carriers. How It Works How It Works 🛒 Purchase the Test: Select and buy the test online. 📧 Receive Instructions: After payment confirmation, receive instructions for sample collection. ✨ Sample Collection: Your veterinarian collects the sample. 📄 Download Submission Form: Download the printable submission form here. 📮 Send Samples: Send to our lab by regular mail or express delivery to:Equigerminal LabRua Eduardo Correia, Nº133030-507 Coimbra, PORTUGAL 📄 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. More Info View More Info For more detailed information on the qPCR Test, including sample collection and submission instructions, please visit our website or contact our support team. Visit our detailed diagnosis page for more information. FAQs View FAQs How does the qPCR test work? The qPCR test detects the genome of Babesia caballi and Theileria equi, providing a highly sensitive and specific method for identifying the presence of the pathogens. What types of samples are required for the test? The qPCR test requires 5 mL of blood collected in a K3 EDTA tube. How long does it take to get the test results? The turnaround time for the qPCR test is typically 2 working days from the receipt of the sample in the laboratory. What should be done if a horse tests positive for Piroplasmosis? Horses that test positive should be isolated to prevent the spread of the disease. Follow biosecurity measures and consult with a veterinarian for appropriate treatment and management. How can Piroplasmosis be prevented? Prevention involves testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals.  

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Equine Piroplasmosis Bundle: cELISA & qPCR Our Equine Piroplasmosis Bundle combines cELISA and qPCR tests for comprehensive detection of antibodies to Babesia caballi and Theileria equi. This bundle is crucial for the official trading, import, and export of horses. Product Overview The bundle includes: Two cELISA Tests: Detect antibodies to Babesia caballi and Theileria equi. Used for official trading and movement of horses. Follows ISO/IEC 17025 standards, ensuring high quality and reliability. qPCR Test: Detects the genome of Babesia caballi and Theileria equi, providing highly sensitive detection of piroplasms, ideal for early infection detection and during febrile peaks. Sample Requirements 5 mL of blood or serum - collect blood in a dry tube for cELISA 5 mL of blood - collect blood in K3 EDTA tube for qPCR Turnaround Time Standard Processing: Results in 2 working days after sample arrival at the laboratory. Clients are responsible for organizing and covering the costs of sending the samples to the laboratory. What is Piroplasmosis? Equine piroplasmosis (EP) is a tick-borne disease of horses caused by the intraerythrocytic protozoan parasites Babesia caballi and Theileria equi. These agents are transmitted through a tick vector. Infected animals may remain carriers of these blood parasites for long periods and act as sources of infection for other ticks. The parasites are found in southern Europe, Asia, countries of the Commonwealth of Independent States, Africa, Cuba, South and Central America, and parts of the southern United States. Clinical Signs Incubation period: 12 to 19 days for T. equi and 10 to 30 days for B. caballi. Per-acute form: Rare, with only clinical observation being moribund or dead animals. Acute form: Fever, reduced appetite, elevated respiratory and pulse rates, congestion of mucous membranes, dark red urine, smaller and drier faecal balls, anaemia, and/or icterus. Subacute form: Similar to acute form but with weight loss and intermittent fever. Mucous membranes may vary from pale pink to bright yellow. Chronic form: Mild inappetence, poor performance, weight loss. Documented case fatality rates vary from 10–50%. Transmission Babesia caballi sporozoites invade red blood cells (RBCs), transforming into trophozoites which divide into merozoites, capable of infecting new RBCs. B. caballi can be found in various organs of tick vectors and transmit transovarially. Theileria equi sporozoites, inoculated into horses via a tick bite, invade lymphocytes, develop into schizonts, and release merozoites that invade RBCs. T. equi develop in the salivary glands of the tick vector and are not transmitted transovarially. Transmission is also possible through mechanical vectors contaminated by infected blood. Prevention Sanitary Prophylaxis: Testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals. Medical Prophylaxis: No current biological products are available. Antiprotozoal agents only temporarily clear T. equi from carriers. How It Works How It Works 🛒 Purchase the Test: Select and buy the test online. 📧 Receive Instructions: After payment confirmation, receive instructions for sample collection. ✨ Sample Collection: Your veterinarian collects the sample. 📄 Download Submission Form: Download the printable submission form here. 📮 Send Samples: Send to our lab by regular mail or express delivery to:Equigerminal LabIPN Incubadora, Rua Pedro Nunes, EdifC3030-199 Coimbra, PORTUGAL 📄 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. More Info View More Info For more detailed information on the cELISA and qPCR Tests, including sample collection and submission instructions, please visit our website or contact our support team. Visit our detailed diagnosis page for more information. FAQs View FAQs How do the cELISA and qPCR tests work? The cELISA test detects antibodies to Babesia caballi and Theileria equi, providing a highly sensitive and specific method for identifying the presence of the pathogens. The qPCR test detects the genome of these pathogens, making it ideal for early infection detection and during febrile peaks. What types of samples are required for the tests? The cELISA test requires 5 mL of blood or serum collected in a dry tube. The qPCR test requires 5 mL of blood collected in a K3 EDTA tube. How long does it take to get the test results? The turnaround time for the cELISA and qPCR tests is typically 2 working days from the receipt of the sample in the laboratory. What should be done if a horse tests positive for Piroplasmosis? Horses that test positive should be isolated to prevent the spread of the disease. Follow biosecurity measures and consult with a veterinarian for appropriate treatment and management. How can Piroplasmosis be prevented? Prevention involves testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals.

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Pathogen profile Screening of 6 pathogens responsible anemia signs in equines : Equine Infectious Anemia Virus (EIAV), Anaplasma phagocytophilum, Borrelia Burgdorferi, Leptospira interrogans, Babesia caballi and Theileria equi. Sample 5 mL of blood - K3 EDTA tube Turnaround time 2 to 5  working days

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 Pathogen test  The RT-qPCR test detects the genome (RNA) of Eastern Equine Encephalitis (EEE) virus. Sample 5 mL - blood - K3 EDTA tube 5mL - liquor (CSF) - sterile tube Turnaround time 2 to 5 working days   What is Eastern Equine Encephalitis? Eastern equine encephalitis (EEE), also referred to as triple E, is a viral illness transmitted to humans and horses by the bite of an infected mosquito. The insects pick up the virus from biting an infected bird.  Clinical signs EEE viruses affect the nervous system, so affected animals will have fever, depression and changes in behaviour. Signs of infection may also include impaired vision, muscle twitches, circling or head pressing behaviours, the inability to swallow, paralysis and convulsions. Horses infected with EEE often do not survive Transmission The virus is transmitted to people and horses by bites from infected mosquitoes and birds during wet, summer months Prevention Vaccines for EEE are available for horses. Measures to control mosquito populations and minimize mosquito exposure will decrease chances of infection.

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  Certified DNA Disorder-Free Lines Ensure your horse's lineage is free from genetic disorders with our comprehensive DNA testing panel. Certify your horse against 10 genetic disorders: SCID, LFS, CA, PSSM1, HYPP, GBED, HERDA, MH, CM, WFFS. Sample Collection Hair Roots: 30 to 40 hair roots. Blood Sample: 5 mL blood in a K3 EDTA tube. Turnaround Time Standard Processing: Results in 5 to 10 working days after sample arrival at the laboratory. Clients organize and cover the costs of sending the samples. Why Test? Our Certified DNA Disorder-Free Lines test helps breeders, purchasers, and studbook certifiers ensure that horses are free from genetic disorders. This guarantees healthier horses, informed breeding decisions, and enhanced peace of mind. Learn More Results Description The DNA test results will be one of the following: n/n: Negative. No affected allele present. n/P1: Positive heterozygous. One mutated allele present. The horse can pass the allele to 50% of its progeny. P1/P1: Positive homozygous. Two mutated alleles present. The horse will pass the allele to 100% of its offspring. Additional Information Polysaccharide Storage Myopathy (PSSM1) is a hereditary muscle disease that affects many breeds. The condition is caused by a mutation in the GYS1 gene, leading to an abnormal accumulation of glycogen in the muscles. This can cause symptoms such as muscle tremors, stiffness, reluctance to move, and excessive sweating. Management of PSSM1 includes dietary changes and regular exercise to help mitigate symptoms. Check our FAQs for more information FAQs Why is genetic testing important for horse breeders? Genetic testing is essential for breeders to make informed breeding decisions and to ensure that their horses do not carry alleles for genetic disorders. This helps in maintaining the health and performance of the breed. What breeds are affected by SCID and LFS? SCID and LFS are commonly found in Arabian horses and breeds influenced by Arabian bloodlines. Testing is crucial for breeding and purchasing decisions to ensure the health of the horses. How prevalent are genetic disorders in certain horse breeds? Genetic disorders can have significant frequencies in specific breeds. For example, HERDA is prevalent in Quarter Horses, while WFFS often affects Warmbloods. Regular testing helps in identifying carriers and making informed decisions. How do genetic disorders impact horse health? Genetic disorders such as SCID, LFS, PSSM1, HYPP, and others can significantly impact the health, performance, and longevity of horses. Early detection through genetic testing allows for better management and breeding practices to ensure healthier future generations. Visit our full FAQ page for more details. How it Works ✨ Purchase the Test: Select and buy the DNA test online. 📄 Receive Instructions: After payment confirmation, receive instructions for hair root collection and a printable submission form. ✂️ Collect Hair Roots: Pluck hair roots, tape them on the submission form, place it in an envelope or sealed plastic bag. 📬 Send Samples: Send to our lab by regular mail or express delivery to: Equigerminal LabRua Eduardo Correia, Nº133030-504 Coimbra, PORTUGAL 📧 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. ♻️ Note: No need for a sample collection kit, enhancing sustainability by reducing waste and plastic use.

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DNA test for the Congenital Myotonia (CM). This test verifies the presence of the recessive cm gene. Sample  30 to 40 hair roots or 5 mL of blood in a K3 EDTA tube Turnaround time 2 to 5  working days Why test? This genetic test helps breeders to identify horses that carrying the cm recessive mutation. Informed choices can be made for breeding selections, and prevent the born of affected foals. Results description The DNA test verifies the presence of the recessive cm gene and presents results as one of the following: N/ - Normal for Congenital Myotonia (CM).  Absence of the affected variant responsible for Congenital Myotonia N/cm - Carrier of Congenital Myotonia (CM). Presence of one copy of the genetic variant causative of Congenital Myotonia. The horse is clinical healthy and can pass the genetic variant responsible for CM to 50% of their progeny when bred. cm/ - Affected by CM. Presence of two copies of the genetic variant causative of Congenital Myotonia. The horse is affected with Congenital Myotonia and will pass genetic variant to 100% of its offspring. Additional information Congenital Myotonia is an inherited neuromuscular disorder characterised by the slow relaxation of muscles after voluntary contraction or electrical stimulation. This disorder has been identified in New Forest ponies and it is caused by an autosomal recessive mutation, which is responsible for the function of chloride ion channels in the skeletal muscle. Carriers of the mutation appear normal, but when two carriers are mated, a 25 percent chance exists that an affected foal will be produced.  Affected foals appear normal at birth. The first symptoms are recurrent episodes of recumbency and difficulty rising to its feet as a result of muscle stiffness. They occur during the first weeks of age and usually increase in the following months. Picking up the limbs is not possible because of the muscle rigidity. The eye-bulb may be retracted due to the myotonia.

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DNA test The DNA test verifies the presence of 2 mutations of the TBX3 gene responsible for Dun dilution and primitive markings.  Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Why test? The DNA test that will provide information for both dun dilution (D) and the primitive markings (nd1, nd2).  Results description D/ - Homozygous for Dun. The basic coat colour will be diluted by Dun and primitive markings expressed. The Dun genetic variant will be passed on 100% of the offspring. D/nd1 - 1 copy of Dun and one copy of nd1. The basic coat colour will be diluted by Dun and primitive markings expressed. Horse can pass on Dun dilution (50%) or primitive markings without dilution (50%). D/nd2 - 1 copy of Dun and one copy of nd2. Horse will have Dun dilution and express primitive markings. The Dun genetic variant will be passed  with a 50% chance to the offspring nd1/nd1 - Homozygous for nd1. The basic coat colour will not be diluted but primitive markings are expressed in varying levels. The primitive markings will be passed on 100% of the offspring. nd1/nd2 - 1 copy of nd1 and one copy of nd2. The basic coat colour will not be diluted but primitive markings are expressed in varying levels. The primitive markings will be passed on 50% of the offspring. nd2/nd2 - Negative for Dun Dilution and primitive markings. Additional information Dun is a dominant dilution gene of equines characterised by lightening of the body color, leaving the head, lower legs, mane and tail undiluted. Dun is also typically characterised by “primitive markings” consisting of a dark dorsal stripe and sometimes leg barring, shoulder stripes and concentric marks on the forehead. Dun is present in many breeds of horses including (but not limited to) Appaloosa, Bashkir Curly, Iberian horse breeds, Icelandic Horse, Mustang, Norwegian Fjord, Paint, Paso Fino, Peruvian Paso, Quarter Horse and several of the pony breeds  

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DNA test The DNA test verifies the presence of the champagne mutation. Champagne  is a coat dilution modifier. Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Why test? Equigerminal offers testing for the dominant champagne gene-mutation. DNA testing may be useful in cases whereby a horse has previously tested negative for cream or silver dilutions, but appears to have a lightened-coat. Testing is also used to determine Homozygosity of the champagne gene.  Results description The DNA test verifies the presence of the champagne mutation and presents results as one of the following: N/ – Non-champagne horse. N/Ch – Positive for dominant champagne gene, possessing one inherited copy. Coat will be diluted accordingly. Will pass champagne gene to approximately 50% of the offspring. Ch/ – Positive for dominant champagne gene, possessing two inherited copies. Coat will be diluted accordingly. Additional information Champagne dilution is caused by a dominant gene, meaning that a horse with a single copy of the Champagne gene will have Champagne characteristics. The Champagne dilution gene lightens a horse’s coat color by diluting the pigment. The specific color produced will depend on the horse’s base color: bay coats to a golden brown, black coats can lighten to a dark brown, and chestnut coats to an apricot or gold. A horse can carry more than one dilution gene which can further affect coat color. Unlike cream dilution, there are no visual differences between a horse with one copy or two copies of Champagne. Although similar to the cream, pearl and dun dilutions, the Champagne gene has certain characteristics that distinguish it from other dilutions. Common characteristics of a Champagne horse include pinkish freckled or mottled skin, a shiny coat that is often slightly darker in the winter, and a hazel eye color. Champagne horses are typically born with a blue eye color that evolves to a hazel or an amber colour and pink skin that becomes darker and more freckled over time, especially around the eyes and muzzle. A homozygous Champagne horse will always pass one copy of the Champagne gene to its foal. Heterozygous horses have a 50% chance of passing the gene on to its foals.

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