Finds out whether your horse carries R309H, the change in the GYS1 gene that causes Type 1 Polysaccharide Storage Myopathy (PSSM1).The report gives the genotype directly as n/n, n/P1 or P1/P1, from hair roots or EDTA blood, at any age. It looks for the GYS1 variant only, and does not detect PSSM2, MIM or any other muscle disorder.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Finds out whether your horse carries Tobiano, the white spotting pattern caused by a large inversion on horse chromosome 3, next to the KIT gene.The report gives the genotype directly as N/N, To/N or To/To, from hair roots or EDTA blood, at any age. It finds the To/To horses, which give Tobiano to every foal but look exactly like horses with one copy.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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One test, one sample: discover your horse's true colours, the ones it carries unseen, and the palette it can pass on to its foals.

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Finds out whether your horse carries PATN1, the main gene that turns a leopard complex horse from a small blanket into a full leopard or a few-spot. The marker read is a single-letter change in the RFWD3 gene.The report gives the genotype directly as N/N, PATN1/N or PATN1/PATN1, from hair roots or EDTA blood, at any age. PATN1 shows only in horses that also carry LP, so solid horses can carry it hidden.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Two DNA tests from one sample: WFFS, the PLOD1 variant c.2032G>A behind Warmblood Fragile Foal Syndrome, and PSSM1, the GYS1 variant R309H behind Type 1 Polysaccharide Storage Myopathy.The report gives both genotypes directly: n/n, n/WFFS or WFFS/WFFS, and n/n, n/P1 or P1/P1. WFFS carriers are healthy but must not be mated together; one PSSM1 copy is enough to make a horse susceptible.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Finds out whether your horse carries Grey, the STX17 variant that makes a horse born with an ordinary coat turn white with age.The report gives the genotype directly as N/N, G/N or G/G, from hair roots or EDTA blood, at any age, so a grey foal can be identified years before its first white hair.Grey horses get skin melanomas far more often than other horses, and two copies of Grey are linked to more severe melanoma than one.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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The qPCR test detects the genome (DNA) of Streptococcus equi subspecies equi, the pathogen responsible for Strangles. Sample Nasopharyngeal swab (dry swab). See AAEP guidelines. Nasal swabs or draining lymph nodes. Guttural pouch washes. Turnaround Time 2 to 5 working days after sample receipt. What is Strangles? A highly contagious upper respiratory infection caused by S. equi. Transmitted via inhalation or contact with contaminated surfaces (e.g., water buckets). Colonizes tonsils/pharynx and causes abscesses in lymph nodes. View More Info For more detailed information, visit our website. How It Works 🛒 Purchase: Buy the test online. 📄 Download Form: Submission Form 📮 Send Samples: Equigerminal, S.A., HIESE, Rua da Quinta do Sobreiro, 25, Quinta Vale do Espinhal, 3230-343 Penela, PORTUGAL. FAQs View FAQs How does the qPCR test work? Detects DNA with high sensitivity and specificity. What to do if positive? Isolate the horse, apply biosecurity, and consult a vet.  

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This is one of the internationally imposed tests in the import/export of live equines or semen/ova and embryos intended for assisted reproduction. PTE029/7 AGID test (Coggins test) to detect antibodies against equine infectious anemia  Equine Infectious Anaemia is a disease listed in the OIE Terrestrial Animal Health Code and countries are obligated to report the occurrence of the disease according to the OIE Code. Sample 5 mL - blood - serum tube Turnaround time 2 to 5 working days   What is Equine Infectious Anemia? Equine infectious anemia is a very old viral disease that affects horses, asses, mules and hinnies worldwide. It is subject to tight controls in the import/export of live equines and their products Clinical signs This infection may have an acute, chronic or sub-clinical (silent) phase. The acute phase characterised by intermittent fever associated with depression, lethargy, increased heart and breathing rates, haemorrhaging, diarrhoea with blood, bleeding wounds that won’t heal, lack of coordination and rapid weight loss. It can also cause petechial haemorrhages of the mucous membranes and general oedema more evident in the legs and jaundice. The chronic phase characterised by recurrent episodes of fever, anaemia and thrombocytopenia (decrease of blood platelets) interspersed with periods of normality. These episodes will be spread out over time. This disease is often fatal during the acute or chronic phase. Should the animal survive the acute and chronic phase, it enters a silent phase with no evident signs of illness for the remainder of its life. In this silent phase the virus persists but the clinical signs are only manifest if the immune system is weakened by another disease, stress or the administration of corticosteroids. Transmission EIA is caused by a lentivirus of the HIV family, the equine infectious anaemia virus. The virus can be passed from one horse to another through fly, or more rarely, mosquito bites, or by direct contact with blood or blood derivative products (serum and/or plasma). Such as, for example, by: sharing objects contaminated with infected blood (needles, branding tools, etc). The virus can also be passed down from mare to foal via the placenta or, more rarely, in the mother’s colostrum or milk. Potentially, the virus can be transmitted by semen. Prevention There is no treatment, cure or vaccine for this infection. Prevention is crucial to avoid it being passed on. Serological tests for EIA must be done for any horse with anaemia and thrombocytopenia of unknown origin. Regular tests must be done on a yearly basis to keep the holding free from EIA. It is advisable to test studs and brood mares every 90 days in the breeding period.    

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Antibody (IgG) test for Babesia caballi and Theileria equi, the causes of equine piroplasmosis – the test asked for international horse movement.Results as a percentage: for each parasite we report the % inhibition measured in your horse's sample, with the 40% cut-off (below 40% = negative, 40% or more = positive).Equigerminal is DGAV Official Laboratory No. 22 and is accredited by IPAC under ISO/IEC 17025.Same-day results: we test every Wednesday and Friday; samples arriving by 12:00 noon are reported by the end of the day.

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Determines your horse's base coat colour, bay, black or chestnut, by analysing two loci: the Extension locus controls pigment production, red and/or black; the Agouti locus controls the distribution of black pigment.

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Reads six variants sold as markers of Muscle Integrity Myopathy (MIM), formerly called PSSM2: P2 (MYOT), P3 (FLNC), P4 (MYOZ3), P8 (PYROXD1), Px (CACNA2D3) and K1 (COL6A3).Before ordering, note that independent studies found P2, P3 and P4 just as often in healthy horses as in horses diagnosed by muscle biopsy, and we found no published horse study of P8, Px or K1. The result is not a diagnosis and does not rule muscle disease in or out.For a horse with muscle problems, the PSSM1 DNA test and a muscle biopsy taken by your veterinarian are the validated routes. Results are issued within 5 to 10 working days of the sample reaching the laboratory.

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Finds out whether your horse carries the PPIB variant G39R, the cause of Hereditary Equine Regional Dermal Asthenia (HERDA), a fragile-skin disease recorded in American Quarter Horses, American Paint Horses and Appaloosas, and heavily concentrated in certain cutting horse lines. Some reports write the same variant G6R.The report gives the genotype directly as n/n, n/HERDA or HERDA/HERDA, from hair roots or EDTA blood, at any age. It identifies carriers, which look completely normal, before breeding, and gives an answer years before an affected horse breaks down under saddle.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Reads two genes linked to skin melanoma in grey horses: Grey (STX17), and Agouti (ASIP), where the black a version has been linked to more severe melanoma in grey Lipizzaners.The report gives both genotypes and ranks a grey horse's genetic melanoma risk from lower to higher. It is a comparison between grey horses, not a percentage chance, and it does not diagnose melanoma.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Finds out whether your horse carries the PLOD1 variant c.2032G>A, the cause of Warmblood Fragile Foal Syndrome, also called Fragile Foal Syndrome (FFS).The report gives the genotype directly as n/n, n/WFFS or WFFS/WFFS, from hair roots or EDTA blood, at any age. It identifies carriers, which look completely normal and cannot be spotted any other way, so that two carriers are never mated together.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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The PCR test detects the genome (DNA) of Burkholderia mallei, the bacteria responsible for Glanders in equines. Sample 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5 working days   What is Glanders? Glanders is a contagious and fatal disease of horses, donkeys, and mules, caused by infection with the bacterium Burkholderia mallei.The pathogen causes nodules and ulcerations in the upper respiratory tract and lungs. A skin form also occurs, known as ‘farcy’. Control of glanders requires testing of suspect clinical cases, screening of apparently normal equids, and elimination of positive reactors. As B. mallei can be transmitted to humans, all infected/contaminated or potentially infected/contaminated material must be handled in a laboratory with appropriate biosafety and biosecurity controls following a biorisk analysis. Glanders is an OIE listed disease as described in the Terrestrial Animal Health Code of the World Organisation for Animal Health (OIE). As indicated in the OIE Terrestrial Animal Health Code any occurrence of glanders must be notified to the OIE. Clinical signs The disease causes nodules and ulcerations in the respiratory tract and lungs in animals. A skin form, known as ‘farcy’, also occurs. Both acute and chronic forms of the disease have been described. Acute forms occur most frequently in donkeys and mules, with high fever and respiratory signs. In horses, glanders generally takes a more chronic course and they may survive for several years. There are four recognised clinical presentations of glanders: nasal, pulmonary, cutaneous and asymptomatic carrier. These different forms of glanders are usually referred to according to the location of the initial infection. The nasal and pulmonary forms tend to be more acute while the cutaneous form is a chronic process. Inflammatory nodules and ulcers develop in the nasal passages and give rise to a sticky yellow discharge. Stellate scarring follows upon healing of the ulcers. The formation of nodular abscesses in the lungs is accompanied by progressive debility, coughing and may also be accompanied by diarrhoea. In the cutaneous form (“farcy’), the lymph vessels are enlarged; nodular abscesses form along their course, which then ulcerate and discharge yellow pus. Nodules are regularly found in the liver and spleen, leading to wasting and death. Transmission The most common source of infection is ingestion of contaminated food or water. Contaminated aerosols (produced by coughing and sneezing), and contaminated fomites brought to the animals via grooming equipment and tack may also be a source of infection. The bacteria can also enter the body through contact with lesions or abrasions of the skin or through mucosa. In this case, a local infection with ulceration may develop spreading to other parts of the body in the course of the disease. Poor husbandry and feeding conditions as well as animal transport can be predisposing factors. Unsanitary conditions and over-crowded stables are risk factors. Prevention To date, no treatment with veterinary drugs is capable to cure the infection. Control of glanders requires early detection and diagnostic testing of suspected clinical cases, screening of apparently normal equids, and elimination of positive cases. For glanders-free countries, there are recommendations on importing equines. An international veterinary certificate is required attesting that the animals showed no clinical signs of glanders and were kept in an exporting country free of the disease for at least 6 months prior to shipment.

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Anti-Müllerian Hormone (AMH)  Sample 5 mL - blood - serum tube  Turnaround time 2 to 5 working days

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Reads the SLC45A2 Cream dilution, variant c.457G>A. What it does to the coat depends both on how many copies the horse inherited and on its base colour.The report gives the genotype directly as N/N, N/Cr or Cr/Cr, from hair roots or EDTA blood, at any age. It finds the smoky black horses that carry one Cream copy and look like ordinary blacks.Run it with the Extension and Agouti tests to know which cream dilute the horse is, since one copy gives a palomino, a buckskin or a smoky black depending on the base colour.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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ACTH levels are seasonal in horses  Increased ACTH levels could indicate Pituitary Pars Intermedia Dysfunction PPID, also known as Equine Cushing’s Disease. For more information about PPID please check the 2021  EEG recommendations on diagnosis and management of pituitary pars intermedia dysfunction (PPID).   Sample requirements 5 mL of blood in EDTA tube Separate the plasma by centrifugation or gravity and freeze plasma at -20ºC (in a regular freezer).  Send freeze plasma to lab ASAP in a refrigerated package.  Turnaround time 2 to 5 working days

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qPCR test: Detects the genome (DNA) of Equine Herpesvirus Type 4 (EHV-4). Molecular detection of EHV-4 by PCR is the most sensitive, specific, and accurate tool to assess the infectivity of an affected horse. Sample 1 nasal or nasopharyngeal swab (see AAEP guidelines) 5 mL K3 EDTA tube (blood) 20g placental or fetal tissue in a sterile flask Turnaround time 2 to 5 working days What is Herpesvirus Type 4? EHV-4 is a global health risk. Clinical presentation alone is often insufficient for diagnosis as signs overlap with other respiratory diseases. Clinical signs Infections are restricted to respiratory tract epithelium and associated lymph nodes. Abortion in pregnant mares is rare with EHV-4 strains. Establishes latent infection in most horses; viral shedding can reactivate during periods of stress. Transmission Spread via aerosolized secretions from coughing horses. Direct horse-to-horse contact or indirect contact via contaminated objects (fomites). Healthy-appearing horses may still shed the virus through nasal secretions. The environment/air around an infected horse can be contaminated with the virus. Prevention Herd elimination is nearly impossible due to the carrier state; focus on disease prevention. Strategy: Prophylactic immunization and preventive herd management practices. Management: Isolate horses in small closed groups and minimize stress. During outbreaks: Disinfect contaminated areas, isolate affected animals, and implement strict biosecurity protocols.

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The PCR test detects the genome (DNA) of  Trypanosoma equiperdum, the pathogen responsible for Dourine. Sample 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5 working days   What is Dourine? Dourine is a chronic or acute contagious disease of breeding equids that is transmitted directly from animal to animal during coitus. The causal organism is Trypanosoma equiperdum. Dourine is the only trypanosomosis that is not transmitted by an invertebrate vector. Trypanosoma equiperdum differs from other trypanosomes in that it is primarily a tissue parasite that is rarely detected in the blood. There is no known natural reservoir of the parasite other than infected equids. Clinical signs Dourine is characterised mainly by swelling of the genitalia, cutaneous plaques and neurological signsThe symptoms vary with the virulence of the strain, the nutritional status of the horse, and stress factors. The clinical signs often develop over weeks or months. They frequently wax and wane; relapses may be precipitated by stress. This can occur several times before the animal either dies or experiences an apparent recovery.Genital edema and a mucopurulent discharge are often the first signs. Mares develop a mucopurulent vaginal discharge, and the vulva becomes oedematous; The genital region, perineum and udder may become depigmented. Abortion can occur with more virulent strains. Stallions develop edema of the prepuce and glans penis, and can have a mucopurulent discharge from the urethra. In stallions, the swelling may spread to the scrotum, perineum, ventral abdomen and thorax. Neurological signs can develop soon after the genital edema, or weeks to months later. Restlessness and weight shifting from one leg to another is often followed by progressive weakness, incoordination and, eventually, paralysis. Facial paralysis, which is generally unilateral, may be seen in some animals. Conjunctivitis and keratitis are common, and in some infected herds, ocular disease may be the first sign of dourine. Anemia and intermittent fever may also be found. In addition, dourine results in a progressive loss of condition, predisposing animals to other diseases. Transmission Unlike other trypanosomal infections, dourine is transmitted almost exclusively during breeding. Transmission from stallions to mares is more common, but mares can also transmit the disease to stallions. T. equiperdum can be found in the vaginal secretions of infected mares and the seminal fluid, mucous exudate of the penis, and sheath of stallions. Periodically, the parasites disappear from the genital tract and the animal becomes noninfectious for weeks to months. Non infectious periods are more common late in the disease. Male donkeys can be asymptomatic carriers. Rarely, infected mares pass the infection to their foals, possibly before birth or through the milk. Infections are also thought to occur through mucous membranes such as the conjunctiva. Other means of transmission may also be possible; however, there is currently no evidence that arthropod vectors play any role in transmission. Sexually immature animals that become infected can transmit the organism when they mature. Prevention To prevent dourine from being introduced into a herd or region, new animals should be quarantined and tested by serology. When dourine is found in an area, quarantines and the cessation of breeding can prevent transmission while infected animals are identified. Dourine can be eradicated from a herd, using serology to identify infected equids. Infected animals are euthanised. In some cases, stallions have been castrated to prevent disease transmission; however, geldings can still transmit the disease if they display copulatory behavior. Successful treatment with trypanocidal drugs has been reported in some endemic areas. However, therapeutic regimes have not been thoroughly investigated, and treatment is usually discouraged due to fears that the organism will persist inapparently. Good hygiene should be used at assisted matings. No vaccine is available.

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Introduction Ordering and utilizing our canine genetic tests is a straightforward process designed to provide you with valuable insights into your dog's health and lineage. Simply follow the instructions provided in the kit to collect a DNA sample from your dog and send it back to our laboratory for analysis. Benefits Verified Parentage: Understand your dog's unique genetic heritage and confirm their parentage, ensuring their pedigree. Verified Identity: Secure a unique genetic ID for your dog, invaluable for breeders, registration, and in case of loss. Procedure Follow the instructions provided in the kit to collect a DNA sample from your dog and send it back to our laboratory for analysis. Results Results are typically available within 2-3 weeks and include a detailed report on the genetic status of your dog. FAQs How do I collect the sample? Follow the instructions provided in the kit to collect a DNA sample. How long does it take to get results? Results are typically available within 2-3 weeks from the time we receive the sample. What should I do if my dog tests positive for a genetic predisposition? If your dog tests positive, consult with your veterinarian to create a management plan tailored to your dog's needs.

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Screening of 5 pathogens responsible for respiratory disease in equines: EHV-1, EHV-4, Equine Influenza, Rhodococcus equi (Pneumonia), and Streptococcus equi (Strangles). Our lab is approved by the FEI for EHV-1 testing. Sample 1 nasopharyngeal swab (see AAEP guidelines) 5 mL blood (K3 EDTA tube) Turnaround time 2 to 5 working days

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Finds out whether your horse carries the RYR1 variant R2454G, the cause of Malignant Hyperthermia (MH) in Quarter Horses and related breeds. The horse looks normal until anaesthesia, hard exercise or stress sets off a fast and potentially fatal attack. Some reports write the same variant R2455G.The report gives the genotype directly as n/n, n/MH or MH/MH, from hair roots or EDTA blood, at any age. Knowing it before surgery lets your veterinarian medicate ahead of anaesthesia. Testing alongside PSSM1 is recommended in Quarter Horse breeds.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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Finds out whether your horse carries the GBE1 variant c.102C>A, the cause of Glycogen Branching Enzyme Deficiency (GBED), a recessive disease of Quarter Horses, Paint Horses and related breeds that is always fatal.The report gives the genotype directly as n/n, n/GBED or GBED/GBED, from hair roots or EDTA blood. It finds the healthy carriers whose mating causes abortions and the loss of newborn foals, and confirms GBED in an affected foal.Results are issued as a DNA certificate within 5 to 10 working days of the sample reaching the laboratory.

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