About the Test – Comprehensive Allergy Panel The Comprehensive Allergy Panel evaluates sensitivity to a wide range of environmental and insect allergens in horses. Using advanced detection technologies, the test measures allergen-specific IgE with high precision and avoids cross-reactivity with IgG or CCDs (cross-reactive carbohydrate determinants). All results are interpreted using the HERBU scoring system, which provides clinically relevant categorization of IgE reactivity levels for each allergen. Why Test? Identifying specific allergens responsible for your horse's symptoms is essential for targeted allergy management. The Comprehensive Allergy Panel enables early detection and supports the development of effective treatment and prevention strategies, including immunotherapy. Benefits of the  Test Broad screening of European environmental and insect allergens. High sensitivity: detects specific IgE levels down to 10 pg per sample. Rapid turnaround: results in 2 to 7 working days. Results expressed according to the HERBU score, ensuring clinically meaningful interpretation. Specific for IgE only, with no interference from IgG or non-relevant cross-reactions. Special Considerations The test should not be performed during or immediately after anti-allergy treatments (e.g., corticosteroids, antihistamines), as these can suppress IgE responses and yield false-negative results. FAQs How can anti-allergy treatments affect the test results? These treatments can lower IgE production or activity, potentially resulting in underestimation of allergen sensitivity. It is advised to wait an appropriate period after treatment before testing. What makes the Comprehensive Allergy Panel different from standard allergy tests?This panel uses IgE-specific detection and a refined scoring system (HERBU) to ensure accurate, clinically significant results. It avoids misleading positives from IgG or carbohydrate-based cross-reactions. Environmental Allergens Screened  Perennial Allergens (Mites and Molds) : Acarus siro, Dermatophagoides farinae,  Dermatophagoides pteronyssinus,  Tyrophagus putrescentiae, Alternaria alternata, Cladosporium herbarum. Seasonal Allergens (Crops, Grasses, Weeds):  Secale cereale, Sorghum halepensis, Poa pratensis, Festuca pratensis. Trees: Betula populifolia, Salix Caprea, Corylus avellana, Fagus sylvatica.   Insects: Culicoides spp,  Culex tarsalis, Tabanus spp  

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Results Results are provided (reactive 0 to 5 ) for all allergens in all 4 allergen classes, with identification of the results obtained for each specific allergen in each class. In total, results are provided for 34 allergens. Pack of 4 full screenings: 1 - Environmental - Mites and Fungi + 2 - Environmental - Pollens + 3 - Environmental - Insects + 4 - Food Sample 5 mL serum or 10 mL - blood - serum tube   Turnaround time 10  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility

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Allergy testing Results: Results are given reactive/ non-reactive with identification of 8 different specific food allergen for both IgE and IgG.  Reactive/Non-reactive IgE and IgG for the following food allergens: wheat, barley, oats, corn, molasses, soy, yeast, alfalfa Sample 2 mL serum or 4 mL - blood - serum tube   Turnaround time 10  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility

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Allergy testing Results: The results are provided as positive/negative results for each of the 6 allergens composed of feathers, hair and skin from 6 species of animals that may cohabit with horses:  1 - cat, 2 - dog, 3 - rabbit, 4 - guinea pig, 5 - parrot, 6 - feather mix Sample 2 mL serum or 4 mL - blood - serum tube   Turnaround time 10  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility  

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Why Test? Equine allergies are relatively common and can affect horses of any breed, age, or sex. Allergic reactions involving the skin—particularly those associated with insect bites—are a major cause of discomfort. Conditions such as Sweet Itch (Insect Bite Hypersensitivity) are diagnosed by excluding other common causes of pruritus and dermatitis. Once confirmed, identifying the specific insect allergens affecting your horse allows for targeted management and improved quality of life. Key Benefits Fast and accurate identification of relevant insect allergens Non-invasive sample collection Not affected by most medications Highly reproducible and standardized procedure Insects Tested Results are provided for each of the following six insect genera, with reactivity scored from 0 to 5 using the HERBU scoring system:   Genus Species Observation Chrysops spp. Includes multiple species of Chrysops (deer flies) Culex spp. Includes multiple mosquito species of Culex Tabanus spp. Includes multiple horse fly species of Tabanus Stomoxys  calcitrans Stable fly: specific to Stomoxys calcitrans Culicoides spp. includes Culicoides nubeculosus Includes multiple species of Culicoides, notably C. nubeculosus (sweet itch in horses) Blattella germanica Cockroach: specific to Blattella germanica (German cockroach)   Allergen-specific IgE is measured, ensuring precise detection with no cross-reactivity from IgG or CCDs. Sample Requirements 3 mL serum or 6 mL whole blood (in serum tube) Turnaround time 2-7  working days  

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Why Test? Seasonal allergic reactions in horses can affect the skin, respiratory tract, and even digestion. Identifying specific sensitivities to pollens — such as grasses, weeds, and trees — is crucial for effective long-term management and avoiding unnecessary treatments. Horses of any breed, age, or sex can be affected, often with symptoms that peak during spring and summer. Key Benefits Precise allergen identification: Based on specific IgE response HERBU scoring system: Results reported on a 0–5 scale for clinical clarity Minimally invasive: Requires only a blood sample — no sedation, shaving, or intradermal injections Highly reproducible: Standardized and validated laboratory method Fast turnaround: Results ready in 2 to 7 working days What’s Tested? The test covers three key allergen classes relevant to seasonal exposures. Results are IgE-specific, avoiding cross-reactivity with IgG or CCDs. 🌱 Grasses and Crops Poa pratensis (Kentucky bluegrass)  Phleum pratense (Timothy grass)  Cynodon dactylon (Bermuda grass)  Festuca pratensis (Meadow fescue)  Sorghum halepense (Johnson grass)  Secale cereale (Rye)  Dactylis glomerata (Orchard grass)  Artemisia vulgaris (Mugwort)  🌿 Weeds Chenopodium album (Lamb’s quarters)  Plantago lanceolata (Plantain)  Rumex crispus (Curly dock)  🌳 Trees Betula populifolia (Gray birch) Salix caprea (Goat willow)  Corylus avellana (Hazel)  Fagus sylvatica (Beech)  🧪 Sample Requirements 2 mL serumor 4 mL whole blood (serum tube) ⏱️ Turnaround Time 2 to 7 working days 🧬 About the Results Results are expressed using the HERBU score (0–600) Each allergen is classified into clinical reactivity classes (0–5) IgE-specific testing avoids cross-reactions with IgG or irrelevant allergens Supports formulation of custom Allergen-Specific Immunotherapy (ASIT) when indicated

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Allergy testing Results are given as Positive or Negative allergy for each of the 4 classes of allergens, without identification of the specific allergen in each class.  Allergens classes: 1 - Dust mites, 2 - Pollens, 3 - Moulds, 4 - Insects Sample 2 mL serum or 4 mL - blood - serum tube   Turnaround time 5  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility  

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Parameter Focuses on the fundamental study of cells, including their structure, function, and essential processes, with an emphasis on counting the total number of cells in a given sample to assess general cellular health. Sample Typically involves basic cell samples from tissues or fluid aspirations, with cell counts performed on simple, unstained samples or using basic staining techniques to facilitate easier observation and counting. Turnaround time Results, including cell counts, for basic cytological examinations are usually ready within 1-2 working days, reflecting the straightforward nature of the analysis.

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Parameter Bacteriological and mycological culture Includes antibiogram (if applied) Sample Aspirates, fluids, solid tissues, swabs. Turnaround time 2-5 days

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Parameter Cytological, bacteriological and mycological analysis Antibiogram (if applied) Sample Equine Bronchoalveolar Lavage (BAL) Turnaround time 3-8 days

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Pathogen test  IFAT  for Borrelia burgdorferi, the pathogen responsible for the Lyme disease or Borreliose. IgG + IgM IgG titers of 1:64 are considered doubtful, IgG titers of 1:128 and higher are considered positive in the sense of an infection that has occurred. Recent infections appear with a positive IgM titer, but may be IgG negative. Sample 5 mL - blood - serum tube Turnaround time 2 to 5 working days   What is Lyme disease? Lyme disease is a tickborne illness that results from infection with members of the Borrelia burgdorferi sensu lato complex. These organisms are maintained in wild animals, but they can affect humans and some species of domesticated animals, like horses. Lyme disease has been reported in North America, Europe, Australia and parts of Asia. Clinical signs Clinical signs appear in less than 10% of horses infected with the bacteria. Signs include: Lameness (usually of larger joints) that shifts from limb to limb; Generalised stiffness; Soreness in the large joints and back; Low-grade fever (which may or may not be present); Behavioural changes such as reluctance to work and irritability; Laminitis (occasionally associated with Lyme disease) Horses do not show a skin rash with Lyme disease. Swelling around a tick bite in a horse is generally due to a reaction to the tick’s saliva, not Lyme disease. Transmission The ticks become infected when they feed on rodents such as the white-footed mouse that carry the bacteria. The tick can then pass on this infection when it feeds on another host, such as a horse or deer. The bacteria migrate from the tick to the horse after 12 to 24 hours of attachment. In areas where the incidence of disease is high among people, only about 50% of horses are likely to become infected. Of these horses, less than 1 in 10 develops clinical signs of the disease. The remaining horses either have subclinical infection (carry the antibodies against the bacteria but remain clinically healthy) or their immune systems fight off the bacteria (and these horses carry the antibodies to Lyme disease for up to a year). People can also be infected with Lyme disease, but there is no risk of the disease being transmitted from horses to humans. Prevention Since there is no vaccine available, prevention is focused on control of the tick population: Perform a daily tick examination. Remember that it takes 12 to 24 hours of attachment for bacteria to migrate from the tick to the host. Treat turned out horses with permethrin-based insect repellents during peak adult deer tick seasons: early spring, late summer, and fall. Minimize habitat for ticks and their hosts.  

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Pathogen test The microscopic agglutination test (MAT) is the reference test method for the serodiagnosis of leptospirosis both in humans and in animals (World Health Organization (WHO), 2003; World Organisation for Animal Health (OIE, 2008). This test detects antibodies to specific serovars using live leptospiral antigens, and can be performed on serum from any species. Sample5 mL - blood - serum tube Turnaround time2 to 5 working days What is Leptospirosis?Leptospirosis is a bacterial infection that can result in abortion, chronic uveitis, and/or kidney failure in horses and can also infect humans, pets, and other livestock. Clinical signsSome horses with leptospirosis appear completely normal. Others might show generalised flulike signs.More serious cases present as mid- to late-term abortions, chronic uveitis (an eye disease that’s the leading cause of blindness in horses), or renal (kidney) disease.Foals from infected dams born alive might suffer from malnutrition, jaundice, pulmonary haemorrhage, or severe respiratory distress—all of which can be fatal. If you can start treatment immediately—before the infection damages eyes or organs—horses with leptospirosis generally have good prognoses. And, you can protect the rest of your herd (and yourself and other animals) by isolating infected horses, treating your other horses with preventive antibiotics or, depending on the species involved, vaccinating. TransmissionLeptospirosis is caused by spiral-shaped bacteria called spirochetes, specifically leptospires, that enter an animal’s body through mucous membranes in areas such as the nostrils, lips, eyes, trachea, stomach, genitals, or anus, or through broken skin. In addition to mammals (horses, humans, squirrels, voles, and scores more), amphibians (such as frogs) and reptiles (including snakes) can become infected with and pass on the disease-causing bacteria. In horses, foals can become infected in utero.Leptospires most commonly live and multiply in the renal tubules (where urine collects in the kidneys) of reservoir or carrier hosts such as rodents, wildlife, and domestic animals. In addition to spreading in urine, leptospires can be transmitted via infected blood or tissues or by infected urine splashing into eyes or the mouth.When an infected reservoir host urinates, the leptospires pass out of its body in the urine and contaminate the surrounding ground and/or water. In fact, exposure to standing water, such as ponds or floodwaters, is the biggest risk factor for leptospirosis infection. Occasionally, says Carter, animals (horses, livestock, etc.) inhale leptospires, ingest them with feed, or transmit them via wounds or bites. PreventionHumans who work with animals or have frequent exposure to them are at a higher risk of contracting leptospirosis.The leptospirosis is now classified as a re-emerging disease. Worldwide, the incidence is increasing, which may be due to increased episodes of flooding. According to the CDC, most human leptospirosis symptoms are flulike and include: high fever; headache; chills; muscle aches; vomiting; jaundice (yellow skin and eyes); red eyes; and abdominal pain.Treatment is fairly straightforward. The emphasis now is on rapid diagnostics so the disease can be diagnosed quickly and treatment started.Prevention is characterised differently for different risk groups. For occupational exposure (for example, veterinarians and animal caretakers that might be exposed to the disease), appropriate PPE (personal protective equipment, including protective gloves and glasses) is important. For recreational exposure, preventing contact of mucous membranes and broken skin with contaminated water is advised.

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Anaplasma phagocytophilum / Erlichiosis test by IFAT the bacterium (formerly known as Ehrlichia phagocytophila and Ehrlichia equi)  responsible for the Equine Anaplasmosis. Sample5 mL of blood in a serum tube Turnaround time2 to 5 working daysWhat is Equine Anaplasmosis?Anaplasmosis is a tick-borne disease caused by the bacterium Anaplasma phagocytophilum that infects white blood cells. The disease is transmitted by ticks. The risk of transmission to people is unclear at this time.Although horses and people appear to be infected with strains of the same bacteria, it is believed that people also acquire the infection from tick bites, and not directly from infected horses. Clinical signsThe severity of signs varies with the age of the animal and duration of the illness. Signs may be mild.Horses less than 1 year old may have a fever only; horses 1 to 3 years old develop fever, depression, mild limb swelling, and lack of coordination.Adults exhibit the characteristic signs of fever, poor appetite, depression, reluctance to move, limb swelling, and jaundice.Fever is highest during the first 1 to 3 days of infection, but may last for 6 to 12 days. Signs become more severe over several days.Any existing infection (such as a leg wound or respiratory infection) can be made worse. TransmissionThe disease is transmitted by ticks.Immature ticks pick up the bacterium from rodents who serve as reservoirs, maintain it as they mature, and then transmit it to the horse they feed off of as adults. It is unknown how long the tick has to be attached before transmission occurs.It takes approximately 2-3 weeks after disease transmission for the horse to develop clinical signs of Anaplasmosis, meaning that by the time signs are noticed the tick is long gone.Phagocytophilum organisms infect neutrophils and eosinophils in the blood. PreventionThe disease is easily treated in the early stages using appropriate antibiotics.The severity of the disease is variable; many horses recover after 14 days without treatment. However, rare fatalities have occurred that are believed to be associated with secondary infections.Horses with severe signs and neurologic signs may benefit from injectable corticosteroids.Recovered horses develop immunity for at least 2 years and are not carriers.Tick control measures are mandatory for control of the disease.There is no vaccine.

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Parameter Copper  Sample 5 mL - blood - serum tube   Turnaround time 2 to 5 working days

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Parameter Culture of the uterus  Sample Uterine swab in special culture transport system Turnaround time 1-15 day for bacterial growth Several days (>15) for yeast and other fungal growth 

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Parameter Entails a detailed examination of cells, using advanced techniques not only to identify abnormalities and diagnose diseases but also to perform precise cell counts, including differential counts to distinguish between different cell types or identify specific cellular abnormalities. Sample Typically involves cell samples from tissues, lavages or fluid aspirations, with cell counts performed using staining techniques to facilitate easier observation and counting, and  to provide detailed counts and information on cell types. Turnaround time The results for advanced cytological studies, including differential cell counts, may take longer than basic cytology due to the complexity of the tests and analyses involved are usually ready within 2-4 working days.

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Pathogen test  The bacterial culture will be reported positive if a beta-hemolytic Streptococcus species is grown and identified specifically as the Strep equi (formal name is Streptococcus equi subspecies equi). Sample Live Strep equi organisms must be recovered by a swab or wash of the nasopharynx, nasal passages, or draining abscesses. Turnaround time 2 to 5 working days   What is Stranglers? Strangles is a highly contagious upper respiratory infection of horses caused by the bacteria Streptococcus equi subspecies equi (S. equi). It is transmitted by inhalation or direct contact with contaminated surfaces (for example horses sharing water buckets). The bacteria colonize the horse’s tonsils and pharynx within hours of infection, and then infect the lymph nodes under and behind the jaw resulting in abscessation of these structures days later. Horses develop a fever initially, but are typically not contagious during the initial 48-72 hours. Rarely, infection spreads to other parts of the body resulting in abscesses in other organs such as the intestines, kidneys, lungs, spleen or liver. This is often called “bastard strangles” or metastatic abscessation. A few horses may develop a hypersensitivity reaction to the bacteria with repeated exposure either in the form of infection or vaccination otherwise known as purpura hemorrhagica. Horses that develop classic clinical signs and are not treated with antibiotics have the potential to develop immune protection up to five years. Clinical signs Classic clinical signs include a fever (often >103°F or 39.5°C) first, followed by one or more of the following symptoms: depression, thick nasal discharge and lymph node enlargement under the jaw and/or in the throat latch region. The abscessed lymph nodes may drain externally or into the guttural pouches (blind-end sacs connected to the throat in horses) resulting in nasal discharge. Horses that have been vaccinated for strangles or horses that have previous partial immunity may develop milder signs of upper respiratory tract infection. Bastard strangles cases may develop colic signs, fever, and/or weight loss with or without a history of previous strangles disease or exposure. Horses with purpura hemorrhagica may develop edema of the head, trunk, and/or legs; and broken blood vessels or bruising of the mucous membranes of the mouth, eyes and nose. Additional signs can include fever, severe depression, and muscle tightness. The severity of symptoms in purpura hemorrhagica cases ranges from mild to life-threatening. Transmission Strangles is caused by oral exposure of a horse to S. equi bacteria. Once within the oral cavity, the bacteria invade the tonsils and subsequently colonize the lymph nodes. Bacteria can be transmitted through contact with pus or nasal discharges from an infected horse, or from contaminated bedding or barn equipment (water troughs, buckets, etc.). Flies may also act as vectors, spreading the bacteria from horse to horse. Under the right conditions, S. equi can survive in the environment for weeks or months. Exposure of a horse to S. equi does not necessarily mean that it will come down with strangles. Factors that influence the risk of disease include dose of bacteria (poor sanitation and direct contact with nasal secretions and pus increase the chance of disease); immune status of horse. Previously exposed horses are often immune to the disease, or do not get as sick as unexposed horses. During the first three to six months of life, foals are often protected by maternal antibodies. Vaccination can also increase resistance to the disease; stress (poor nutrition, overcrowding, lengthy transportation or pre-existing diseases increase the risk of strangles). Strangles may be transmitted by “silent shedders” who do not display signs of disease. These horses commonly carry the strangles organism in the guttural pouch, an air sac at the back of the horse’s throat. Detection of these animals requires guttural pouch endoscopy (passing an endoscope via the horse’s nose into the guttual pouch). Strangles is most commonly transmitted by acutely ill or recovering horses that are still shedding bacteria in their nasal secretions. Bacterial culture results have a turnaround time of 2 to 3 days. The DNA test known as Polymerase Chain Reaction (PCR) takes less than a day. However, it may take an additional 1 to 2 days to send samples to the laboratory. Prevention Biosecurity on the farm is necessary to prevent spread of disease. Isolate new horses for three weeks prior to introducing them to the rest of the population. Isolate any horse with a fever and signs of strangles. Do not share tack or equipment between sick horses and others Perform twice daily monitoring of rectal temperatures of all horses in an outbreak to identify new cases. Stop all movement of horses to and from farm when strangles is identified. Disinfect water buckets daily. Use strict hygiene between horses to reduce spread of the disease. Ideally, three throat flush samples are obtained from recovering horses and any horses who were in contact with sick horses at approximately weekly intervals and tested for S. equi subsp equi by PCR and culture. Identification of strangles bacteria in clinically recovered horses may mean the guttural pouches have retained some infection. Endoscopy of the guttural pouches provides visualisation of any pus or dried debris (chondroids) that harbor the bacteria. A small number of horses will recover from strangles and continue to shed bacteria from the guttural pouch, causing recurrent farm outbreaks. Detection and treatment of these “silent carriers” (S. equi bacteria in guttural pouches) via endoscopy and PCR is essential for preventing disease recurrence on a farm. Discuss vaccination types and recommendations with your veterinarian. Vaccination does not provide 100% immunity against S. equi infection. Vaccination is not recommended during or within two years of a strangles outbreak due to the increased risk of purpura hemorrhagica.

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Shopping for someone else but not sure what to give them? Is he/she a Horse-lover ? Give them the gift of choice with a Equigerminal gift card. Gift cards are delivered by email and contain instructions to redeem them at checkout.Our gift cards have no additional processing fees.

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Equine Piroplasmosis Bundle: cELISA & qPCR Our Equine Piroplasmosis Bundle combines cELISA and qPCR tests for comprehensive detection of antibodies to Babesia caballi and Theileria equi. This bundle is crucial for the official trading, import, and export of horses. Product Overview The bundle includes: Two cELISA Tests: Detect antibodies to Babesia caballi and Theileria equi. Used for official trading and movement of horses. Follows ISO/IEC 17025 standards, ensuring high quality and reliability. qPCR Test: Detects the genome of Babesia caballi and Theileria equi, providing highly sensitive detection of piroplasms, ideal for early infection detection and during febrile peaks. Sample Requirements 5 mL of blood or serum - collect blood in a dry tube for cELISA 5 mL of blood - collect blood in K3 EDTA tube for qPCR Turnaround Time Standard Processing: Results in 2 working days after sample arrival at the laboratory. Clients are responsible for organizing and covering the costs of sending the samples to the laboratory. What is Piroplasmosis? Equine piroplasmosis (EP) is a tick-borne disease of horses caused by the intraerythrocytic protozoan parasites Babesia caballi and Theileria equi. These agents are transmitted through a tick vector. Infected animals may remain carriers of these blood parasites for long periods and act as sources of infection for other ticks. The parasites are found in southern Europe, Asia, countries of the Commonwealth of Independent States, Africa, Cuba, South and Central America, and parts of the southern United States. Clinical Signs Incubation period: 12 to 19 days for T. equi and 10 to 30 days for B. caballi. Per-acute form: Rare, with only clinical observation being moribund or dead animals. Acute form: Fever, reduced appetite, elevated respiratory and pulse rates, congestion of mucous membranes, dark red urine, smaller and drier faecal balls, anaemia, and/or icterus. Subacute form: Similar to acute form but with weight loss and intermittent fever. Mucous membranes may vary from pale pink to bright yellow. Chronic form: Mild inappetence, poor performance, weight loss. Documented case fatality rates vary from 10–50%. Transmission Babesia caballi sporozoites invade red blood cells (RBCs), transforming into trophozoites which divide into merozoites, capable of infecting new RBCs. B. caballi can be found in various organs of tick vectors and transmit transovarially. Theileria equi sporozoites, inoculated into horses via a tick bite, invade lymphocytes, develop into schizonts, and release merozoites that invade RBCs. T. equi develop in the salivary glands of the tick vector and are not transmitted transovarially. Transmission is also possible through mechanical vectors contaminated by infected blood. Prevention Sanitary Prophylaxis: Testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals. Medical Prophylaxis: No current biological products are available. Antiprotozoal agents only temporarily clear T. equi from carriers. How It Works How It Works 🛒 Purchase the Test: Select and buy the test online. 📧 Receive Instructions: After payment confirmation, receive instructions for sample collection. ✨ Sample Collection: Your veterinarian collects the sample. 📄 Download Submission Form: Download the printable submission form here. 📮 Send Samples: Send to our lab by regular mail or express delivery to:Equigerminal LabIPN Incubadora, Rua Pedro Nunes, EdifC3030-199 Coimbra, PORTUGAL 📄 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. More Info View More Info For more detailed information on the cELISA and qPCR Tests, including sample collection and submission instructions, please visit our website or contact our support team. Visit our detailed diagnosis page for more information. FAQs View FAQs How do the cELISA and qPCR tests work? The cELISA test detects antibodies to Babesia caballi and Theileria equi, providing a highly sensitive and specific method for identifying the presence of the pathogens. The qPCR test detects the genome of these pathogens, making it ideal for early infection detection and during febrile peaks. What types of samples are required for the tests? The cELISA test requires 5 mL of blood or serum collected in a dry tube. The qPCR test requires 5 mL of blood collected in a K3 EDTA tube. How long does it take to get the test results? The turnaround time for the cELISA and qPCR tests is typically 2 working days from the receipt of the sample in the laboratory. What should be done if a horse tests positive for Piroplasmosis? Horses that test positive should be isolated to prevent the spread of the disease. Follow biosecurity measures and consult with a veterinarian for appropriate treatment and management. How can Piroplasmosis be prevented? Prevention involves testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals.

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Pathogen profile Screening of 6 pathogens responsible anemia signs in equines : Equine Infectious Anemia Virus (EIAV), Anaplasma phagocytophilum, Borrelia Burgdorferi, Leptospira interrogans, Babesia caballi and Theileria equi. Sample 5 mL of blood - K3 EDTA tube Turnaround time 2 to 5  working days

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Pathogen profile This profile includes 10 qPCR tests that detects 10 pathogens responsible for  neurological disease in horses such as: EHV-1, EPM, EEE, EEV, VEE, JEV, WEE, WNV, RBV, Hendra. Sample 5 mL - blood ( K3 EDTA tube) and/or 5mL - liquor (CSF) in a sterile tube Turnaround time 2 to 5  working days  

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 Pathogen test  The RT-qPCR test detects the genome (RNA) of Eastern Equine Encephalitis (EEE) virus. Sample 5 mL - blood - K3 EDTA tube 5mL - liquor (CSF) - sterile tube Turnaround time 2 to 5 working days   What is Eastern Equine Encephalitis? Eastern equine encephalitis (EEE), also referred to as triple E, is a viral illness transmitted to humans and horses by the bite of an infected mosquito. The insects pick up the virus from biting an infected bird.  Clinical signs EEE viruses affect the nervous system, so affected animals will have fever, depression and changes in behaviour. Signs of infection may also include impaired vision, muscle twitches, circling or head pressing behaviours, the inability to swallow, paralysis and convulsions. Horses infected with EEE often do not survive Transmission The virus is transmitted to people and horses by bites from infected mosquitoes and birds during wet, summer months Prevention Vaccines for EEE are available for horses. Measures to control mosquito populations and minimize mosquito exposure will decrease chances of infection.

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  DNA test kit DNA test to predict the genetic potential for being a small, medium or tall height horse. Sample 30 to 40 hair roots - envelope or 5 mL of blood  - K3 EDTA tube Turnaround time 5 to 8  working days Why test?  Confidently predict the expected mature height at withers of a young foal. Identify horses that are 70% likely to be within the specific height range desired by the owner. Produce horses of a desired height more consistently. Results description The DNA test verifies the presence of the (C) allele associated to tall height horses, and presents the results as one of the following:  C/C – Tall physical height. This genotype, with two copies of the (C) allele,  is associated with large horses, (169+/-4 cm height at the withers). This genotype can be found in draught horses, Westphalians (31%), Oldenburgs (29%), Hanoverians (28%), Holsteiners (18%) and Pureblood Lusitanos (4%). T/C – Medium physical height. This genotype, with one copy of the (T) allele and another of the (C) allele,  is associated with medium-sized horses (164+/-5 cm height at the withers). This genotype can be found in the Holsteiner (64%), Westphalian (60%), Hanoverian (56%) and Pureblood Lusitano (29%). T/T – Small physical height. This genotype, with two copies of the (T) allele, is associated with smaller horses (159 +/- 4 cm height at the withers) and ponies (<148 cm height at the withers). This genotype can be found in the majority of ponies, Arabs (100%) and Pureblood Lusitanos (67%).                                 Additional information The development of the skeletal apparatus is regulated by the expression of the gene LCORL. Recent studies have identified a variation of a simple nucleotide base (Single Nucleotide Polymorphism) in the promoter of this gene. The replacement of a thymine (T) with a cytosine (C) varies the expression of the LCORL gene. This variation in the expression influences the horse’s dimension. Consequently, if the expression of the LCORL gene is diminished the resulting horse has a larger body dimension (bones are longer).

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