112 products

  • Allergy test to feathers, hair and skin of other animals - Equigerminal

    Allergy test to feathers, hair and skin of other animals

    Allergy testing Results: The results are provided as positive/negative results for each of the 6 allergens composed of feathers, hair and skin from 6 species of animals that may cohabit with horses:  1 - cat, 2 - dog, 3 - rabbit, 4 - guinea pig, 5 - parrot, 6 - feather mix Sample 2 mL serum or 4 mL - blood - serum tube   Turnaround time 10  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility  

    €179.00

  • Allergy test to Insects - Equigerminal

    Sweet Itch Allergy Testing

    Why Test? Equine allergies are relatively common and can affect horses of any breed, age, or sex. Allergic reactions involving the skin—particularly those associated with insect bites—are a major cause of discomfort. Conditions such as Sweet Itch (Insect Bite Hypersensitivity) are diagnosed by excluding other common causes of pruritus and dermatitis. Once confirmed, identifying the specific insect allergens affecting your horse allows for targeted management and improved quality of life. Key Benefits Fast and accurate identification of relevant insect allergens Non-invasive sample collection Not affected by most medications Highly reproducible and standardized procedure Insects Tested Results are provided for each of the following six insect genera, with reactivity scored from 0 to 5 using the HERBU scoring system:   Genus Species Observation Chrysops spp. Includes multiple species of Chrysops (deer flies) Culex spp. Includes multiple mosquito species of Culex Tabanus spp. Includes multiple horse fly species of Tabanus Stomoxys  calcitrans Stable fly: specific to Stomoxys calcitrans Culicoides spp. includes Culicoides nubeculosus Includes multiple species of Culicoides, notably C. nubeculosus (sweet itch in horses) Blattella germanica Cockroach: specific to Blattella germanica (German cockroach)   Allergen-specific IgE is measured, ensuring precise detection with no cross-reactivity from IgG or CCDs. Sample Requirements 3 mL serum or 6 mL whole blood (in serum tube) Turnaround time 2-7  working days  

    €229.86

  • Allergy test to Pollens/ Seasonal - Equigerminal

    Seasonal Allergy Testing – Pollen Sensitivities in Horses

    Why Test? Seasonal allergic reactions in horses can affect the skin, respiratory tract, and even digestion. Identifying specific sensitivities to pollens — such as grasses, weeds, and trees — is crucial for effective long-term management and avoiding unnecessary treatments. Horses of any breed, age, or sex can be affected, often with symptoms that peak during spring and summer. Key Benefits Precise allergen identification: Based on specific IgE response HERBU scoring system: Results reported on a 0–5 scale for clinical clarity Minimally invasive: Requires only a blood sample — no sedation, shaving, or intradermal injections Highly reproducible: Standardized and validated laboratory method Fast turnaround: Results ready in 2 to 7 working days What’s Tested? The test covers three key allergen classes relevant to seasonal exposures. Results are IgE-specific, avoiding cross-reactivity with IgG or CCDs. 🌱 Grasses and Crops Poa pratensis (Kentucky bluegrass)  Phleum pratense (Timothy grass)  Cynodon dactylon (Bermuda grass)  Festuca pratensis (Meadow fescue)  Sorghum halepense (Johnson grass)  Secale cereale (Rye)  Dactylis glomerata (Orchard grass)  Artemisia vulgaris (Mugwort)  🌿 Weeds Chenopodium album (Lamb’s quarters)  Plantago lanceolata (Plantain)  Rumex crispus (Curly dock)  🌳 Trees Betula populifolia (Gray birch) Salix caprea (Goat willow)  Corylus avellana (Hazel)  Fagus sylvatica (Beech)  🧪 Sample Requirements 2 mL serumor 4 mL whole blood (serum tube) ⏱️ Turnaround Time 2 to 7 working days 🧬 About the Results Results are expressed using the HERBU score (0–600) Each allergen is classified into clinical reactivity classes (0–5) IgE-specific testing avoids cross-reactions with IgG or irrelevant allergens Supports formulation of custom Allergen-Specific Immunotherapy (ASIT) when indicated

    €249.00

  • Basic allergy screening - Environmental & Insect - Equigerminal

    Basic allergy screening - Environmental & Insect

    Allergy testing Results are given as Positive or Negative allergy for each of the 4 classes of allergens, without identification of the specific allergen in each class.  Allergens classes: 1 - Dust mites, 2 - Pollens, 3 - Moulds, 4 - Insects Sample 2 mL serum or 4 mL - blood - serum tube   Turnaround time 5  working days   Why test? Equine allergies are common and can affect any breed, age or sex of horse. Symptoms involving the skin, respiratory and gastrointestinal systems can occur for a number of reasons with the diagnosis of allergy being made by systematically ruling out other common conditions. Once diagnosed, knowing what allergens your horse is sensitive to allows you to manage their condition in a way that is specific to their individual needs. Key points: Rapid and easy identification of potential offending allergens Non-invasive and not influenced by most medications Standardised procedure with excellent reproducibility  

    €175.00

  • SARS-CoV-2 molecular testing for Cats - Equigerminal

    Feline COVID19 Molecular

    Protect your pets from SARS-CoV-2 infection Cats are susceptible to SARS-CoV-2 by reverse zoonosis. If you're infected with COVID-19, you can infect your pets.Our team confirmed in a seroepidemiologic survey* of cats and dogs in Portugal and found antibodies against SARS-CoV-2 in 15/69 (21.74%) cats and 7/148 (4.73%) dogs. By performing this test will allow you to know if your Cat is infected with SARS-CoV-2. This test detects viral RNA of SARS-CoV-2 by RTqPCR. Please note that viral RNA is detectable only in the first two weeks after infection.  Molecular test  Reference laboratory diagnostic tests for SARS-CoV-2 are carried out by means of genome amplification tests (RNA) in a polymerase chain reaction in real time, preceded by reverse transcription (rRT-PCR). Sample type Exudate from the nasopharyngeal and oropharyngeal collected through a swab and/or rectal swabs.   Turnaround time Standard processing - Results in 2-5 working days after sample arrival at the laboratory. Clients organise and support the costs of sending the samples to the laboratory. PREMIUM processing - Results in 24 hours after sample arrival. Includes free express delivery** . The laboratory organises Express shipping with pick-up of the package at the client's address and delivery at the laboratory. ** PREMIUM SERVICES INCLUDE AN EXPRESS SHIPPING DELIVERY FOR EUROPEAN COUNTRIES FROM NON REMOTE REGIONS. Check here to know if you are in a remote European region. For remote regions EXTRA fees are applied.    FAQs Why should we test cats ? The test should be done in cats who have had contact with confirmed cases of COVID-19. There are confirmed cases of infections of cats by SARS-CoV-2 through transmission of the virus by humans or other infected cats.   For more info check  the OIE website When cats should take the test? In general, the test can be performed 5 to 14 days after contact with confirmed cases of COVID-19.   The test it is based on the detection of the SARS-CoV-2 genome (RNA) by real-time rRT-PCR techniques in exudates from the nasopharynx, oropharynx and rectal swabs. In dogs the viral load is higher in the first days of infection and starts to decrease after 15 days after infection.   What is the specificity and sensitivity of these tests? The real-time RT-PCR test used is 100% specific and does not cross-react with other animal coronaviruses. For molecular diagnosis, we use two different SARS-CoV-2 genes as targets.The sensitivity of the molecular test depends on the viral load present in the sample. The viral load is maximal 5-7 days after infection and from then on it starts to decrease, becoming diminished 15 days after infection.   References *Barroso, R., Vieira-Pires, A., Antunes, A., & Fidalgo-Carvalho, I. (2022). Susceptibility of Pets to SARS-CoV-2 Infection: Lessons from a Seroepidemiologic Survey of Cats and Dogs in Portugal. Microorganisms, 10(2), 345. https://doi.org/10.3390/microorganisms10020345

    €65.00

  • SARS-CoV-2 molecular testing for Dogs - Equigerminal

    Canine COVID19 Molecular

    Protect your pets from SARS-CoV-2 infection Dogs are susceptible to SARS-CoV-2 by reverse zoonosis. If you're infected with COVID-19, you can infect your pets. Our team confirmed in a seroepidemiologic survey* of cats and dogs in Portugal and found antibodies against SARS-CoV-2 in 15/69 (21.74%) cats and 7/148 (4.73%) dogs. By performing this test will allow you to know if your Dog is infected with SARS-CoV-2. This test detects viral RNA of SARS-CoV-2 by RTqPCR. Please note that viral RNA is detectable only in the first two weeks after infection.  Molecular test  Reference laboratory diagnostic tests for SARS-CoV-2 are carried out by means of genome amplification tests (RNA) in a polymerase chain reaction in real time, preceded by reverse transcription (rRT-PCR).   Sample type Exudate from the nasopharyngeal and oropharyngeal collected through a swab and/or rectal swabs.   Turnaround time Standard processing - Results in 2-5 working days after sample arrival at the laboratory. Clients organise and support the costs of sending the samples to the laboratory. PREMIUM processing - Results in 24 hours after sample arrival. Includes free express delivery** . The laboratory organises Express shipping with pick-up of the package at the client's address and delivery at the laboratory. ** PREMIUM SERVICES INCLUDE AN EXPRESS SHIPPING DELIVERY FOR EUROPEAN COUNTRIES FROM NON REMOTE REGIONS. Check here to know if you are in a remote European region. For remote regions EXTRA fees are applied.    Timeline of the SARS-CoV-2 infection in a Dog in a COVID19+ household.     From the diagnosis of COVID-19 in the dog owners (22 January 2021) to the first and second positive RT-qPCR test of the dog on 29 January 2021 and 4 February 2021, respectively, and the positive ELISA test on 14 February 2021, 16 days after the first positive RT-qPCR test. The green color means positive serological or molecular result and the red color means a negative serological or molecular result. The date corresponds to the day of the blood or swab sampling from the dog.   FAQs Why should we test dogs ? The test should be done in dogs who have had contact with confirmed cases of COVID-19. There are confirmed cases of infections of dogs by SARS-CoV-2 through transmission of the virus by humans. However, the viral load retrieved in dogs is considered low. For more info check the OIE website When dogs should take the test? In general, the test can be performed 5 to 14 days after contact with confirmed cases of COVID-19.   The test it is based on the detection of the SARS-CoV-2 genome (RNA) by real-time rRT-PCR techniques in exudates from the nasopharynx and oropharynx. In human the viral load is higher in the first days of infection and starts to decrease after 15 days after infection. What is the specificity and sensitivity of these tests? The real-time RT-PCR test used is 100% specific and does not cross-react with other human coronaviruses. For molecular diagnosis, we use two different SARS-CoV-2 proteins as targets.The sensitivity of the molecular test depends on the viral load present in the sample. The viral load is maximal 5-7 days after infection and from then on it starts to decrease, becoming diminished 15 days after infection. References *Barroso, R., Vieira-Pires, A., Antunes, A., & Fidalgo-Carvalho, I. (2022). Susceptibility of Pets to SARS-CoV-2 Infection: Lessons from a Seroepidemiologic Survey of Cats and Dogs in Portugal. Microorganisms, 10(2), 345. https://doi.org/10.3390/microorganisms10020345

    €65.00

  • SARS-CoV-2 antibody testing for Cats - Equigerminal

    Feline COVID19 Antibody

    Protect your pets from SARS-CoV-2 infection Cats are susceptible to SARS-CoV-2 by a reverse zoonosis. If you have COVID-19 you can infect your pets. Our team confirmed this finding in a seroepidemiologic survey* of cats and dogs in Portugal and found antibodies against SARS-CoV-2 in 15/69 (21.74%) cats and 7/148 (4.73%) dogs.  For more information check our peer reviewed publication at micro-organism (MDPI).  As in people, animals often have mild clinical signs, or are asymptomatic, but there are also reports of fatal cases.  Why testing By performing this test will allow you to know if your Cat has been infected with SARS-CoV-2. This test detects IgG antibodies against SARS-CoV-2 Nucleoprotein (NP)  and Spike (S) protein.  Please note, that IgG antibodies arise 2-3 weeks after infection and are maintained for some months.    Sample  2 mL blood in a dry tube or 1 mL of sera. Turnaround time  Standard processing - Results in 2-5 working days after sample arrival at the laboratory. Clients organise and support the costs of sending the samples to the laboratory. PREMIUM processing - Results in 5 hours after sample arrival. Includes free express delivery** . The laboratory organises Express shipping with pick-up of the package at the client's address and delivery at the laboratory. ** PREMIUM SERVICES INCLUDE AN EXPRESS SHIPPING DELIVERY FOR EUROPEAN COUNTRIES FROM NON REMOTE REGIONS. Check here to know if you are in a remote European region. For remote regions EXTRA fees are applied.    References *Barroso, R., Vieira-Pires, A., Antunes, A., & Fidalgo-Carvalho, I. (2022). Susceptibility of Pets to SARS-CoV-2 Infection: Lessons from a Seroepidemiologic Survey of Cats and Dogs in Portugal. Microorganisms, 10(2), 345.  https://doi.org/10.3390/microorganisms10020345  Main conclusions of the study:  - Cats that lived in COVID-19 positive households are at more risk to be infected with SARS-CoV-2  and some cats can become ill or may die.  - Cat to cat transmission of SARS-CoV-2 is also a possibility. Cat that lived in COVID-19 negative households but lived in free range and contact with other cats also tested positive.  - If you test COVID-19 positive please avoid direct contact with your cat, or ware a mask.  - Serological testing should be performed  2-3 weeks after contact with a COVID-19 positive case.   - One to 2 weeks after contact with a COVID-19 positive case a molecular RT-qPCR is recommended. Please check more info about molecular tests for cats here .  For more information please check the coverage article of our scientific publication written by an independent professional. If you would like to collaborate and participate in our study, please contact us at support@equigerminal.pt to find out if you qualify for the study and receive free testing for SARS-CoV-2.  

    €30.00

  • SARS-CoV-2 antibody testing for Dogs - Equigerminal

    Canine COVID19 Antibody

    Protect your pets from SARS-CoV-2 infection Dogs are susceptible to SARS-CoV-2 by a reverse zoonosis. If you have COVID-19 you can infect your pets. Our team confirmed this finding in a seroepidemiologic survey* of cats and dogs in Portugal and found antibodies against SARS-CoV-2 in 15/69 (21.74%) cats and 7/148 (4.73%) dogs.  By performing this test will allow you to know if your Dog has been infected with SARS-CoV-2. This test detects IgG antibodies against SARS-CoV-2 Nucleoprotein (NP)  and Spike (S) protein.  Please note, that IgG antibodies arise 2-3 weeks after infection and are maintained for some months.  As in people, animals often have mild clinical signs, or are asymptomatic, but there are also reports of fatal cases.  Sample  2 mL blood in a dry tube or 1 mL of sera. Turnaround time  Standard processing - Results in 2-5 working days after sample arrival at the laboratory. Clients organise and support the costs of sending the samples to the laboratory. PREMIUM processing - Results in 5 hours after sample arrival. Includes free express delivery** . The laboratory organises Express shipping with pick-up of the package at the client's address and delivery at the laboratory. ** PREMIUM SERVICES INCLUDE AN EXPRESS SHIPPING DELIVERY FOR EUROPEAN COUNTRIES FROM NON REMOTE REGIONS. Check here to know if you are in a remote European region. For remote regions EXTRA fees are applied.    References *Barroso, R., Vieira-Pires, A., Antunes, A., & Fidalgo-Carvalho, I. (2022). Susceptibility of Pets to SARS-CoV-2 Infection: Lessons from a Seroepidemiologic Survey of Cats and Dogs in Portugal. Microorganisms, 10(2), 345. https://doi.org/10.3390/microorganisms10020345

    €30.00

  • Gift Card EG - Equigerminal

    Gift Card EG

    Shopping for someone else but not sure what to give them? Is he/she a Horse-lover ? Give them the gift of choice with a Equigerminal gift card. Gift cards are delivered by email and contain instructions to redeem them at checkout.Our gift cards have no additional processing fees.

    €50.00 - €200.00

  • Equine Piroplasmosis qPCR

    Equine Piroplasmosis qPCR

    Our Equine Piroplasmosis qPCR Test provides accurate detection of the genome of Babesia caballi and Theileria equi, the primary agents responsible for Equine Piroplasmosis. This test is ideal for early infection detection and during febrile peaks. Product Overview qPCR Test: Detects the genome of Babesia caballi and Theileria equi, providing highly sensitive detection of piroplasms. Sample Requirements 5 mL of blood - collect blood in K3 EDTA tube for qPCR Turnaround Time Standard Processing: Results in 2 working days after sample arrival at the laboratory. Clients are responsible for organizing and covering the costs of sending the samples to the laboratory. What is Piroplasmosis? Equine piroplasmosis (EP) is a tick-borne disease of horses caused by the intraerythrocytic protozoan parasites Babesia caballi and Theileria equi. These agents are transmitted through a tick vector. Infected animals may remain carriers of these blood parasites for long periods and act as sources of infection for other ticks. The parasites are found in southern Europe, Asia, countries of the Commonwealth of Independent States, Africa, Cuba, South and Central America, and parts of the southern United States. Clinical Signs Incubation period: 12 to 19 days for T. equi and 10 to 30 days for B. caballi. Per-acute form: Rare, with only clinical observation being moribund or dead animals. Acute form: Fever, reduced appetite, elevated respiratory and pulse rates, congestion of mucous membranes, dark red urine, smaller and drier faecal balls, anaemia, and/or icterus. Subacute form: Similar to acute form but with weight loss and intermittent fever. Mucous membranes may vary from pale pink to bright yellow. Chronic form: Mild inappetence, poor performance, weight loss. Documented case fatality rates vary from 10–50%. Transmission Babesia caballi sporozoites invade red blood cells (RBCs), transforming into trophozoites which divide into merozoites, capable of infecting new RBCs. B. caballi can be found in various organs of tick vectors and transmit transovarially. Theileria equi sporozoites, inoculated into horses via a tick bite, invade lymphocytes, develop into schizonts, and release merozoites that invade RBCs. T. equi develop in the salivary glands of the tick vector and are not transmitted transovarially. Transmission is also possible through mechanical vectors contaminated by infected blood. Prevention Sanitary Prophylaxis: Testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals. Medical Prophylaxis: No current biological products are available. Antiprotozoal agents only temporarily clear T. equi from carriers. How It Works How It Works 🛒 Purchase the Test: Select and buy the test online. 📧 Receive Instructions: After payment confirmation, receive instructions for sample collection. ✨ Sample Collection: Your veterinarian collects the sample. 📄 Download Submission Form: Download the printable submission form here. 📮 Send Samples: Send to our lab by regular mail or express delivery to:Equigerminal LabRua Eduardo Correia, Nº133030-507 Coimbra, PORTUGAL 📄 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. More Info View More Info For more detailed information on the qPCR Test, including sample collection and submission instructions, please visit our website or contact our support team. Visit our detailed diagnosis page for more information. FAQs View FAQs How does the qPCR test work? The qPCR test detects the genome of Babesia caballi and Theileria equi, providing a highly sensitive and specific method for identifying the presence of the pathogens. What types of samples are required for the test? The qPCR test requires 5 mL of blood collected in a K3 EDTA tube. How long does it take to get the test results? The turnaround time for the qPCR test is typically 2 working days from the receipt of the sample in the laboratory. What should be done if a horse tests positive for Piroplasmosis? Horses that test positive should be isolated to prevent the spread of the disease. Follow biosecurity measures and consult with a veterinarian for appropriate treatment and management. How can Piroplasmosis be prevented? Prevention involves testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals.  

    €86.10

  • Equine Piroplasmosis Bundle: cELISA & qPCR Equine Piroplasmosis Bundle: cELISA & qPCR

    Equine Piroplasmosis Bundle: cELISA & qPCR

    Equine Piroplasmosis Bundle: cELISA & qPCR Our Equine Piroplasmosis Bundle combines cELISA and qPCR tests for comprehensive detection of antibodies to Babesia caballi and Theileria equi. This bundle is crucial for the official trading, import, and export of horses. Product Overview The bundle includes: Two cELISA Tests: Detect antibodies to Babesia caballi and Theileria equi. Used for official trading and movement of horses. Follows ISO/IEC 17025 standards, ensuring high quality and reliability. qPCR Test: Detects the genome of Babesia caballi and Theileria equi, providing highly sensitive detection of piroplasms, ideal for early infection detection and during febrile peaks. Sample Requirements 5 mL of blood or serum - collect blood in a dry tube for cELISA 5 mL of blood - collect blood in K3 EDTA tube for qPCR Turnaround Time Standard Processing: Results in 2 working days after sample arrival at the laboratory. Clients are responsible for organizing and covering the costs of sending the samples to the laboratory. What is Piroplasmosis? Equine piroplasmosis (EP) is a tick-borne disease of horses caused by the intraerythrocytic protozoan parasites Babesia caballi and Theileria equi. These agents are transmitted through a tick vector. Infected animals may remain carriers of these blood parasites for long periods and act as sources of infection for other ticks. The parasites are found in southern Europe, Asia, countries of the Commonwealth of Independent States, Africa, Cuba, South and Central America, and parts of the southern United States. Clinical Signs Incubation period: 12 to 19 days for T. equi and 10 to 30 days for B. caballi. Per-acute form: Rare, with only clinical observation being moribund or dead animals. Acute form: Fever, reduced appetite, elevated respiratory and pulse rates, congestion of mucous membranes, dark red urine, smaller and drier faecal balls, anaemia, and/or icterus. Subacute form: Similar to acute form but with weight loss and intermittent fever. Mucous membranes may vary from pale pink to bright yellow. Chronic form: Mild inappetence, poor performance, weight loss. Documented case fatality rates vary from 10–50%. Transmission Babesia caballi sporozoites invade red blood cells (RBCs), transforming into trophozoites which divide into merozoites, capable of infecting new RBCs. B. caballi can be found in various organs of tick vectors and transmit transovarially. Theileria equi sporozoites, inoculated into horses via a tick bite, invade lymphocytes, develop into schizonts, and release merozoites that invade RBCs. T. equi develop in the salivary glands of the tick vector and are not transmitted transovarially. Transmission is also possible through mechanical vectors contaminated by infected blood. Prevention Sanitary Prophylaxis: Testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals. Medical Prophylaxis: No current biological products are available. Antiprotozoal agents only temporarily clear T. equi from carriers. How It Works How It Works 🛒 Purchase the Test: Select and buy the test online. 📧 Receive Instructions: After payment confirmation, receive instructions for sample collection. ✨ Sample Collection: Your veterinarian collects the sample. 📄 Download Submission Form: Download the printable submission form here. 📮 Send Samples: Send to our lab by regular mail or express delivery to:Equigerminal LabIPN Incubadora, Rua Pedro Nunes, EdifC3030-199 Coimbra, PORTUGAL 📄 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. More Info View More Info For more detailed information on the cELISA and qPCR Tests, including sample collection and submission instructions, please visit our website or contact our support team. Visit our detailed diagnosis page for more information. FAQs View FAQs How do the cELISA and qPCR tests work? The cELISA test detects antibodies to Babesia caballi and Theileria equi, providing a highly sensitive and specific method for identifying the presence of the pathogens. The qPCR test detects the genome of these pathogens, making it ideal for early infection detection and during febrile peaks. What types of samples are required for the tests? The cELISA test requires 5 mL of blood or serum collected in a dry tube. The qPCR test requires 5 mL of blood collected in a K3 EDTA tube. How long does it take to get the test results? The turnaround time for the cELISA and qPCR tests is typically 2 working days from the receipt of the sample in the laboratory. What should be done if a horse tests positive for Piroplasmosis? Horses that test positive should be isolated to prevent the spread of the disease. Follow biosecurity measures and consult with a veterinarian for appropriate treatment and management. How can Piroplasmosis be prevented? Prevention involves testing and controlling tick exposure, using repellents, acaricides, and regular inspections, controlling and eradicating the tick vector, and quarantining EP-positive animals.

    €150.00

  • Anemia - Pathogen screening panel - Equigerminal

    Equine Anemia Pathogen Screening Panel

    Pathogen profile Screening of 6 pathogens responsible anemia signs in equines : Equine Infectious Anemia Virus (EIAV), Anaplasma phagocytophilum, Borrelia Burgdorferi, Leptospira interrogans, Babesia caballi and Theileria equi. Sample 5 mL of blood - K3 EDTA tube Turnaround time 2 to 5  working days

    €175.00

  • Neurological signs profile - Equigerminal

    Neurological signs profile

    Pathogen profile This profile includes 10 qPCR tests that detects 10 pathogens responsible for  neurological disease in horses such as: EHV-1, EPM, EEE, EEV, VEE, JEV, WEE, WNV, RBV, Hendra. Sample 5 mL - blood ( K3 EDTA tube) and/or 5mL - liquor (CSF) in a sterile tube Turnaround time 2 to 5  working days  

    €369.00

  • Eastern Equine Encephalitis, RT-qPCR - Equigerminal Eastern Equine Encephalitis, RT-qPCR - Equigerminal

    Eastern Equine Encephalitis, RT-qPCR

     Pathogen test  The RT-qPCR test detects the genome (RNA) of Eastern Equine Encephalitis (EEE) virus. Sample 5 mL - blood - K3 EDTA tube 5mL - liquor (CSF) - sterile tube Turnaround time 2 to 5 working days   What is Eastern Equine Encephalitis? Eastern equine encephalitis (EEE), also referred to as triple E, is a viral illness transmitted to humans and horses by the bite of an infected mosquito. The insects pick up the virus from biting an infected bird.  Clinical signs EEE viruses affect the nervous system, so affected animals will have fever, depression and changes in behaviour. Signs of infection may also include impaired vision, muscle twitches, circling or head pressing behaviours, the inability to swallow, paralysis and convulsions. Horses infected with EEE often do not survive Transmission The virus is transmitted to people and horses by bites from infected mosquitoes and birds during wet, summer months Prevention Vaccines for EEE are available for horses. Measures to control mosquito populations and minimize mosquito exposure will decrease chances of infection.

    €61.50

  • Projected Height test - LCORL/NCAPG - Equigerminal Projected Height test - LCORL/NCAPG - Equigerminal

    Projected Height test, LCORL/NCAPG

      DNA test kit DNA test to predict the genetic potential for being a small, medium or tall height horse. Sample 30 to 40 hair roots - envelope or 5 mL of blood  - K3 EDTA tube Turnaround time 5 to 8  working days Why test?  Confidently predict the expected mature height at withers of a young foal. Identify horses that are 70% likely to be within the specific height range desired by the owner. Produce horses of a desired height more consistently. Results description The DNA test verifies the presence of the (C) allele associated to tall height horses, and presents the results as one of the following:  C/C – Tall physical height. This genotype, with two copies of the (C) allele,  is associated with large horses, (169+/-4 cm height at the withers). This genotype can be found in draught horses, Westphalians (31%), Oldenburgs (29%), Hanoverians (28%), Holsteiners (18%) and Pureblood Lusitanos (4%). T/C – Medium physical height. This genotype, with one copy of the (T) allele and another of the (C) allele,  is associated with medium-sized horses (164+/-5 cm height at the withers). This genotype can be found in the Holsteiner (64%), Westphalian (60%), Hanoverian (56%) and Pureblood Lusitano (29%). T/T – Small physical height. This genotype, with two copies of the (T) allele, is associated with smaller horses (159 +/- 4 cm height at the withers) and ponies (<148 cm height at the withers). This genotype can be found in the majority of ponies, Arabs (100%) and Pureblood Lusitanos (67%).                                 Additional information The development of the skeletal apparatus is regulated by the expression of the gene LCORL. Recent studies have identified a variation of a simple nucleotide base (Single Nucleotide Polymorphism) in the promoter of this gene. The replacement of a thymine (T) with a cytosine (C) varies the expression of the LCORL gene. This variation in the expression influences the horse’s dimension. Consequently, if the expression of the LCORL gene is diminished the resulting horse has a larger body dimension (bones are longer).

    €62.73

  • New Equine Virus (NEV) Viral Load test - Equigerminal

    New Equine Virus (NEV) Viral Load

    Pathogen test  This test determines the NEV viral load of your horse by a molecular test that identifies the NEV genome in circulating blood. This test doesn’t determine the NEV status of your horse. An  undetectable viral load doesn't mean that your horse is free of infection.  Sample 5 mL - blood - K3 EDTA tube or 5 mL - Liquor (CSF). Turnaround time 5 to 10 working days Knowing the NEV status and viral load of your horse can help keep your horse - and others - safe Key points The New Equine Virus (NEV) is a horse lentivirus distinctive from Swamp fever virus (EIAV) and similar to HIV-1. Like in HIV infected humans NEV attacks the immune system and natural defence against illness.  A horse infected with NEV will get weaker and weaker until it can no longer fight off life threatening infections and diseases. The rate at which NEV progresses varies depending on age, general health and genetic background.    Learn more about NEV Explore results If NEV viral load is undetectable - No risk of transmitting NEV An undetectable viral load means that the NEV level in the blood is too low to be detected by a viral load test. NEV positive horses can show undetectable viral loads. Horses with NEV who maintain and undetectable viral load have effectively no risk of transmitting NEV to NEV  negative horses.  If NEV viral load is detectable - Risk of transmitting NEV A detectable viral load means that the NEV level in the blood is high to be detected by a viral load test. Horses with NEV who maintain and detectable viral load have effectively a risk of transmitting NEV to NEV  negative horses.    Take Action - Find the suggested next steps based on results. If your horse has a NEV detectable viral load begin by talking to your veterinarian about therapies to boost the immune system of your horse as well about antiretroviral therapy (ART). Monitoring of NEV viral load levels is crucial to evaluate disease progression and risk.  Like with HIV, ART can’t cure NEV, but can help your horse to live a longer and healthier life. The main goal of ART is to reduce your horse’s viral load to an undetectable level. Learn more about ART here

    €61.50

  • New Equine Virus (NEV) Status & Viral load - Equigerminal

    New Equine Virus (NEV) Status & Viral load

    Pathogen test   This diagnostic profile determines the NEV status of a horse, as well the transmitting with a viral load test.  Includes a serological test for NEV - to determine NEV status and a molecular test to determine the NEV viral load.  Sample 5 mL - blood - serum tube and 5 mL - blood - K3 EDTA tube or 5 mL - Liquor (CSF). Turnaround time 5 to 10 working days   Knowing the NEV status and viral load of your horse can help keep your horse - and others - safe Key points The New Equine Virus (NEV) is a horse lentivirus distinctive from Swamp fever virus (EIAV) and similar to HIV-1. Like in HIV infected humans NEV attacks the immune system and natural defence against illness.  A horse infected with NEV will get weaker and weaker until it can no longer fight off life threatening infections and diseases. The rate at which NEV progresses varies depending on age, general health and genetic background.  Learn more about NEV  Explore results If your horse is NEV negative : Testing shows that your horse doesn’t have NEV. Continue taking steps to keep your horse safe from getting NEV  If your horse is NEV positive : Testing shows that your horse does have NEV, but you can still take steps to protect your horse´s health. NEV viral load test indicates the transmitting risk. An undetectable viral load means that the NEV level in the blood is too low to be detected by a viral load test. Horses with NEV who maintain and undetectable viral load have effectively no risk of transmitting NEV to NEV  negative horses.  Learn more about NEV viral load Take action - Find the suggested next steps based on results If your horse is NEV positive Begin by talking to your veterinarian about therapies to boost the immune system of your horse as well about antiretroviral therapy (ART). Monitoring of NEV viral load levels is crucial to evaluate disease progression and risk.  Like with HIV, ART can’t cure NEV, but can help your horse to live a longer and healthier life. The main goal of ART is to reduce your horse’s viral load to an undetectable level. Learn more about ART here.

    €200.00

  • DNA Disorder-Free Certified

    DNA Disorder-Free Certified

      Certified DNA Disorder-Free Lines Ensure your horse's lineage is free from genetic disorders with our comprehensive DNA testing panel. Certify your horse against 10 genetic disorders: SCID, LFS, CA, PSSM1, HYPP, GBED, HERDA, MH, CM, WFFS. Sample Collection Hair Roots: 30 to 40 hair roots. Blood Sample: 5 mL blood in a K3 EDTA tube. Turnaround Time Standard Processing: Results in 5 to 10 working days after sample arrival at the laboratory. Clients organize and cover the costs of sending the samples. Why Test? Our Certified DNA Disorder-Free Lines test helps breeders, purchasers, and studbook certifiers ensure that horses are free from genetic disorders. This guarantees healthier horses, informed breeding decisions, and enhanced peace of mind. Learn More Results Description The DNA test results will be one of the following: n/n: Negative. No affected allele present. n/P1: Positive heterozygous. One mutated allele present. The horse can pass the allele to 50% of its progeny. P1/P1: Positive homozygous. Two mutated alleles present. The horse will pass the allele to 100% of its offspring. Additional Information Polysaccharide Storage Myopathy (PSSM1) is a hereditary muscle disease that affects many breeds. The condition is caused by a mutation in the GYS1 gene, leading to an abnormal accumulation of glycogen in the muscles. This can cause symptoms such as muscle tremors, stiffness, reluctance to move, and excessive sweating. Management of PSSM1 includes dietary changes and regular exercise to help mitigate symptoms. Check our FAQs for more information FAQs Why is genetic testing important for horse breeders? Genetic testing is essential for breeders to make informed breeding decisions and to ensure that their horses do not carry alleles for genetic disorders. This helps in maintaining the health and performance of the breed. What breeds are affected by SCID and LFS? SCID and LFS are commonly found in Arabian horses and breeds influenced by Arabian bloodlines. Testing is crucial for breeding and purchasing decisions to ensure the health of the horses. How prevalent are genetic disorders in certain horse breeds? Genetic disorders can have significant frequencies in specific breeds. For example, HERDA is prevalent in Quarter Horses, while WFFS often affects Warmbloods. Regular testing helps in identifying carriers and making informed decisions. How do genetic disorders impact horse health? Genetic disorders such as SCID, LFS, PSSM1, HYPP, and others can significantly impact the health, performance, and longevity of horses. Early detection through genetic testing allows for better management and breeding practices to ensure healthier future generations. Visit our full FAQ page for more details. How it Works ✨ Purchase the Test: Select and buy the DNA test online. 📄 Receive Instructions: After payment confirmation, receive instructions for hair root collection and a printable submission form. ✂️ Collect Hair Roots: Pluck hair roots, tape them on the submission form, place it in an envelope or sealed plastic bag. 📬 Send Samples: Send to our lab by regular mail or express delivery to: Equigerminal LabRua Eduardo Correia, Nº133030-504 Coimbra, PORTUGAL 📧 Receive Results: Get the result certificate by email. If you need assistance, contact us at support@equigerminal.pt. ♻️ Note: No need for a sample collection kit, enhancing sustainability by reducing waste and plastic use.

    €299.00

  • Congenital Myotonia - CM - Equigerminal

    Congenital Myotonia - CM

    DNA test for the Congenital Myotonia (CM). This test verifies the presence of the recessive cm gene. Sample  30 to 40 hair roots or 5 mL of blood in a K3 EDTA tube Turnaround time 2 to 5  working days Why test? This genetic test helps breeders to identify horses that carrying the cm recessive mutation. Informed choices can be made for breeding selections, and prevent the born of affected foals. Results description The DNA test verifies the presence of the recessive cm gene and presents results as one of the following: N/ - Normal for Congenital Myotonia (CM).  Absence of the affected variant responsible for Congenital Myotonia N/cm - Carrier of Congenital Myotonia (CM). Presence of one copy of the genetic variant causative of Congenital Myotonia. The horse is clinical healthy and can pass the genetic variant responsible for CM to 50% of their progeny when bred. cm/ - Affected by CM. Presence of two copies of the genetic variant causative of Congenital Myotonia. The horse is affected with Congenital Myotonia and will pass genetic variant to 100% of its offspring. Additional information Congenital Myotonia is an inherited neuromuscular disorder characterised by the slow relaxation of muscles after voluntary contraction or electrical stimulation. This disorder has been identified in New Forest ponies and it is caused by an autosomal recessive mutation, which is responsible for the function of chloride ion channels in the skeletal muscle. Carriers of the mutation appear normal, but when two carriers are mated, a 25 percent chance exists that an affected foal will be produced.  Affected foals appear normal at birth. The first symptoms are recurrent episodes of recumbency and difficulty rising to its feet as a result of muscle stiffness. They occur during the first weeks of age and usually increase in the following months. Picking up the limbs is not possible because of the muscle rigidity. The eye-bulb may be retracted due to the myotonia.

    €43.05

  • Severe Combined Immunodeficiency - SCID - Equigerminal

    SCID Severe Combined Immunodeficiency

    DNA test DNA test for the Severe Combined Immunodeficiency (SCID). SCID is an inherited disease seen in pure and part-bred Arab horses. Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Why test? The DNA test for SCID helps breeders to identify the animals that are carriers of the SCID mutation. This information allows breeders to prevent two carriers from breeding, which reduce the chances of producing an SCID foal. Continued breeding of horses that are carriers of the SCID gene is now possible without the worry of producing SCID foals. For example, carrier stallions that possess highly desirable traits can now be selectively bred to clear (homozygous normal) mares (and vice versa). The resulting foals would have an equal chance of being a carrier or clear of SCID, but would definitely not be affected. The foals could be tested anytime after birth to determine their SCID genotype and future matings could be rationally planned. Results description The DNA test verifies the presence recessive SCID mutation and presents results as one of the following: nn – Non-carrier of the SCID gene.Tested negative for the SCID mutation. nSCID – Heterozygous horse for SCID gene, both the normal and SCID alleles were detected. The horse is a carrier of SCID genetic disorder and there is a 50% chance this horse will pass a SCID allele to its offspring SCID SCID – Carrier of two copies of the SCID gene. Homozygous horse for SCID mutation. The horse is affected with the SCID genetic disorder. Additional information Severe Combined Immunodeficiency Disease (SCID) is an inherited disease seen in pure and part-bred Arab horses. Animals with this inherited condition have an enhanced susceptibility to infection and first show signs of disease at between two days and eight weeks of age. Clinical diagnosis of the disease is not straightforward as the symptoms, such as raised temperature, respiratory complications and diarrhoea, are typical of new-born foals with a range of infections. Foals affected by SCID always die from the disorder within the first six months of life. This happens regardless of the level of veterinary care. SCID is therefore a distressing condition for the effected animal and the owners or caregivers, and results in financial loss due to dead foals and veterinary expenses. The disorder is recessive, which means that a horse must be homozygous positive or have two copies of the defective gene to suffer from the disease. Consequently both the sire and the dam must possess at least one copy of the mutated gene in order for the offspring to be afflicted. Offspring born with one copy of the defective gene and one non-defective copy are considered a carrier and have a 50% chance of passing the defective gene on. A number of studies have attempted to estimate the frequency of SCID carriers in the Arab horse population. Most sources speculate that the percentage of Arab foals which die of SCID is 2-3%. If breeding is random then it would imply that roughly 28-35% of Arab horses are carriers. However, most breeding is rather selective, making the true frequency of carriers in the population somewhat unclear.

    €43.05

  • Dun dilution - Equigerminal

    Dun dilution

    DNA test The DNA test verifies the presence of 2 mutations of the TBX3 gene responsible for Dun dilution and primitive markings.  Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Why test? The DNA test that will provide information for both dun dilution (D) and the primitive markings (nd1, nd2).  Results description D/ - Homozygous for Dun. The basic coat colour will be diluted by Dun and primitive markings expressed. The Dun genetic variant will be passed on 100% of the offspring. D/nd1 - 1 copy of Dun and one copy of nd1. The basic coat colour will be diluted by Dun and primitive markings expressed. Horse can pass on Dun dilution (50%) or primitive markings without dilution (50%). D/nd2 - 1 copy of Dun and one copy of nd2. Horse will have Dun dilution and express primitive markings. The Dun genetic variant will be passed  with a 50% chance to the offspring nd1/nd1 - Homozygous for nd1. The basic coat colour will not be diluted but primitive markings are expressed in varying levels. The primitive markings will be passed on 100% of the offspring. nd1/nd2 - 1 copy of nd1 and one copy of nd2. The basic coat colour will not be diluted but primitive markings are expressed in varying levels. The primitive markings will be passed on 50% of the offspring. nd2/nd2 - Negative for Dun Dilution and primitive markings. Additional information Dun is a dominant dilution gene of equines characterised by lightening of the body color, leaving the head, lower legs, mane and tail undiluted. Dun is also typically characterised by “primitive markings” consisting of a dark dorsal stripe and sometimes leg barring, shoulder stripes and concentric marks on the forehead. Dun is present in many breeds of horses including (but not limited to) Appaloosa, Bashkir Curly, Iberian horse breeds, Icelandic Horse, Mustang, Norwegian Fjord, Paint, Paso Fino, Peruvian Paso, Quarter Horse and several of the pony breeds  

    €43.05

  • Champagne dilution - Equigerminal

    Champagne dilution

    DNA test The DNA test verifies the presence of the champagne mutation. Champagne  is a coat dilution modifier. Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Why test? Equigerminal offers testing for the dominant champagne gene-mutation. DNA testing may be useful in cases whereby a horse has previously tested negative for cream or silver dilutions, but appears to have a lightened-coat. Testing is also used to determine Homozygosity of the champagne gene.  Results description The DNA test verifies the presence of the champagne mutation and presents results as one of the following: N/ – Non-champagne horse. N/Ch – Positive for dominant champagne gene, possessing one inherited copy. Coat will be diluted accordingly. Will pass champagne gene to approximately 50% of the offspring. Ch/ – Positive for dominant champagne gene, possessing two inherited copies. Coat will be diluted accordingly. Additional information Champagne dilution is caused by a dominant gene, meaning that a horse with a single copy of the Champagne gene will have Champagne characteristics. The Champagne dilution gene lightens a horse’s coat color by diluting the pigment. The specific color produced will depend on the horse’s base color: bay coats to a golden brown, black coats can lighten to a dark brown, and chestnut coats to an apricot or gold. A horse can carry more than one dilution gene which can further affect coat color. Unlike cream dilution, there are no visual differences between a horse with one copy or two copies of Champagne. Although similar to the cream, pearl and dun dilutions, the Champagne gene has certain characteristics that distinguish it from other dilutions. Common characteristics of a Champagne horse include pinkish freckled or mottled skin, a shiny coat that is often slightly darker in the winter, and a hazel eye color. Champagne horses are typically born with a blue eye color that evolves to a hazel or an amber colour and pink skin that becomes darker and more freckled over time, especially around the eyes and muzzle. A homozygous Champagne horse will always pass one copy of the Champagne gene to its foal. Heterozygous horses have a 50% chance of passing the gene on to its foals.

    €43.05

  • Silver dilution (Dapple) / MCOA - Equigerminal

    Silver dilution (Dapple)

    DNA test The genetic test verifies the presence of the Silver coat dilution modifier. The Silver genetic variant is associated with Multiple Congenital Ocular Abnormalities (MCOA) in some breeds.   Sample 30 to 40 - hair roots - envelope or 5 mL - blood - K3 EDTA tube Turnaround time 2 to 5  working days Results description The DNA test verifies the presence of the silver gene and presents results as one of the following: N/ – Negative for Silver - No evidence of the genetic variant for Silver. No risk to develop Multiple Congenital Ocular Abnormalities (MCOA) associated to Silver. Z/N - Heterozygous for Silver - The Black and Bay basic coat colour will be diluted by Silver. Black-based horses will be chocolate with flaxen mane and tail. Bay-based horses will have pigment on lower legs lightened and flaxen mane and tail. No effect on chestnut color. Moderate risk to develop MCOA. Z/ – Homozygous for Silver - Two copies of altered sequence detected. Black-based horses will be chocolate with flaxen mane and tail. Bay-based horses will have pigment on lower legs lightened and flaxen mane and tail. No effect on chestnut color, but will pass the variant on to 100% of offspring.  Higher risk to develop severe MCOA. Additional information The Silver dilution behaves as a coat colour dominant trait on bay and black base coat colours. While chestnut base colour is not affected by the Silver dilution and can pass the variant silently to the offspring.  In short, the Silver dilution variant (Z) will only affect coat colour phenotype of black pigmented horses (E/e or E/E) and has no effect on red pigmented horses (e/e).  In addition, the eye disorders associated to Silver genetic variant are incomplete autosomal dominant:  homozygous horses (with two copies of Z)  may be at higher risk of developing severe Multiple Congenital Ocular Abnormalities (MCOA), while heterozygous (with one copy of Z) may develop a milder form of MCOA.   The effects of the silver dilution on coat colour gene can vary widely. The agouti gene affects the coat colour by controlling the distribution of the black pigment whereas the Silver dilution variant dilutes areas of the black pigment. Dilution by the Silver variant on a horse with a uniform black base typically involves lightening of the mane and tail and a dilution of the body to a chocolate color, often dappled as well. A Bay horse carrying the Silver gene will usually have a lightened mane and tail, as well as lightened lower legs. It is important to know that although a red horse (e/e) will not be diluted by the silver variant, it can be a carrier of the genetic variant and thus potentially pass the gene on to its offspring. Silver dilution has been identified in a number of horse breeds including the Quarter horse, the Rocky Mountain horse, the Icelandic horse, Morgans, Shetland ponies and the Miniature horse. References: Brunberg, E., Andersson, L., Cothran, G., Sandberg, K., Mikko, S., Lindgren, G.: A missense mutation in PMEL17 is associated with the silver coat color in the horse. BMC Genetics 7:46, 2006. Andersson, L.S., Wilbe, M., Viluma, A., Cothran, G., Ekesten, B., Ewart, S., Lindgren, G.: Equine Multiple Congenital Ocular Anomalies and Silver Coat Colour Result from the Pleiotropic Effects of Mutant PMEL. PLoS One 8:e75639, 2013.

    €43.05

  • Pearl dilution - Equigerminal

    Pearl dilution

    DNA test The DNA test is designed to verify the presence of the pearl allele (Prl), a coat color dilution modifier discovered in horses of Iberian origin. This variant produces dilutions of the base color, introducing golden tones to the coat.   Sample requirements  20 to 30 hair roots, or 5 mL of blood in a K3 EDTA tube.   Turnaround time The results are available within 2 to 5 working days. Why test? Purpose of the Test Pearl is a rare variant that dilutes the base coat colors in a less pronounced manner than the cream variant (Cr). It can complement the effect of the Cream variant, leading to very diluted coats similar to Cream double dilutes when both are present in heterozygosity. Testing is crucial for breeding purposes, as heterozygous Pearl horses can produce diluted offspring when bred with another Pearl carrier or a Cream dilute horse. The impact of the Pearl dilution varies based on the horse's base color, affecting the phenotype differently across different base colors.    Interpretation of Results for the Pearl Locus  N/N - Negative for Pearl The horse is genetically negative for the pearl allele, meaning it does not have any copies of this genetic variant. Its phenotype reflects the natural, unaltered base coat color. This horse will not pass the pearl dilution trait to its offspring, ensuring the continuation of the base coat color in the lineage. N/Prl – Positive Heterozygous  The horse is positive for the Pearl allele in a heterozygous state, indicating it carries one copy of the pearl variant. This configuration subtly dilutes the base coat color, infusing it with golden tones, although in some instances, the dilution effect may not be visually apparent.  As a heterozygous carrier, there's a 50% probability that it will transmit this dilution trait to its offspring, potentially leading to varied coat colors among the progeny. Prl/Prl -  Positive Homozygous  The horse is positive for the pearl allele in a homozygous state, carrying two copies of this genetic variant. This genotype manifests in a more noticeable dilution of the coat color, even in the absence of other dilution genes. Being homozygous, the horse will invariably pass the pearl allele to all of its offspring, ensuring the trait's propagation and contributing to the diversity of coat colors in future generations.   Additional insights The interplay between the Cream and Pearl genes subtly yet significantly affects horse coat colors, particularly evident in horses heterozygous for both genes (N/Cr + N/Prl). These horses often resemble double cream dilutes but can be distinguished by slightly darker eye colors and a marginally darker coat. Unlike double cream dilutes, the combined dilution effect of heterozygous Cream and Pearl genes might not be as pronounced, requiring careful observation or genetic testing for accurate identification.Homozygous Pearl horses (Prl/Prl) exhibit a more noticeable dilution, displaying pronounced golden tones in their coats compared to their homozygous Cream counterparts (Cr/Cr), whose phenotype is lighter. Interestingly, the eye and skin colors in foals—typically blue and pinkish, respectively—tend to darken with age, while the coat lightens.The subtle dilution effects of a single Pearl allele (N/Prl) often go undetected without genetic analysis, as they minimally alter the horse's appearance. However, the presence of two Pearl alleles (Prl/Prl) significantly enhances the dilution, affecting not just the coat but also the eye color, with amber or green hues depending on the base coat color.Identified in Iberian breeds like the Purebred Lusitano (PSL) and Purebred Spanish Horse (PRE), and speculated in the Spanish Mustang, the Pearl gene's inclusion in genetic discussions highlights its broad impact across equine breeds. This genetic diversity, particularly when Pearl intersects with Cream, underscores the complexity of equine coat colors and the value of genetic testing for breeders. 

    €43.05

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